1
40
4
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1152/jappl.1994.76.2.783" target="_blank" rel="noreferrer noopener">http://doi.org/10.1152/jappl.1994.76.2.783</a>
Pages
783–786
Issue
2
Volume
76
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Daily exercise improved blood pressure homeostasis of rats subjected to surgical stress.
Publisher
An entity responsible for making the resource available
Journal of applied physiology (Bethesda, Md. : 1985)
Date
A point or period of time associated with an event in the lifecycle of the resource
1994
1994-02
Subject
The topic of the resource
*Blood Pressure; *Homeostasis; *Physical Conditioning; Animal; Animals; Female; Male; Operative/*adverse effects; Physiological/*etiology/*physiopathology; Rats; Running; Sprague-Dawley; Stress; Surgical Procedures
Creator
An entity primarily responsible for making the resource
Scislo T J; DiCarlo S E; Jarjoura D G
Description
An account of the resource
The effect of daily spontaneous running on blood pressure homeostasis (BPH) was evaluated in 19 male and 13 female control rats and 7 male and 13 female daily spontaneous running rats subjected to surgery and subsequent repetitive hemodynamic disturbances. BPH was operationally defined as the ability to maintain mean arterial pressure above 60 mmHg during the experimental protocol. The length of time the rats maintained BPH was compared across males and females and trained and control groups. Significant sex (P = 0.01) and training (P = 0.05) effects were found. Females maintained homeostasis longer than males and trained longer than controls. Sex effects were not due to differences in the body mass. The mechanisms responsible for the higher resistance to deterioration of homeostasis merit further investigation.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1152/jappl.1994.76.2.783" target="_blank" rel="noreferrer noopener">10.1152/jappl.1994.76.2.783</a>
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
*Blood Pressure
*Homeostasis
*Physical Conditioning
1994
Animal
Animals
DiCarlo S E
Female
Jarjoura D G
Journal of applied physiology (Bethesda, Md. : 1985)
Male
Operative/*adverse effects
Physiological/*etiology/*physiopathology
Rats
Running
Scislo T J
Sprague-Dawley
Stress
Surgical Procedures
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1152/jappl.1989.67.1.305" target="_blank" rel="noreferrer noopener">http://doi.org/10.1152/jappl.1989.67.1.305</a>
Pages
305–310
Issue
1
Volume
67
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Effect of elevated vascular pressure transients on protein permeability in the lung.
Publisher
An entity responsible for making the resource available
Journal of applied physiology (Bethesda, Md. : 1985)
Date
A point or period of time associated with an event in the lifecycle of the resource
1989
1989-07
Subject
The topic of the resource
*Blood Pressure; Animals; Blood Proteins/*metabolism; Dogs; Female; Hypertension/physiopathology; In Vitro Techniques; Lung/metabolism/*physiopathology; Male; Pulmonary Edema/*etiology/metabolism/physiopathology
Creator
An entity primarily responsible for making the resource
Maron M B
Description
An account of the resource
Neurogenic pulmonary edema (NPE) may develop in individuals with head trauma or seizures and is generally thought to have a hydrostatic basis in the severe degree of pulmonary hypertension that occurs. Recently, it has been suggested that vascular pressures may rise to levels that damage the vessels, leaving the patient at risk for further edema development. The objective of this study was to determine if pulmonary vascular protein permeability is increased in a canine isolated perfused left lower lung lobe (LLL) preparation by pressure transients that may occur in NPE. Venous pressure (Pv) was transiently raised to values ranging from 8 to 102 Torr in 19 LLL. One Pv transient was studied per LLL. After Pv was returned to normal, the osmotic reflection coefficient (sigma d) for total proteins was determined by the hematocrit-protein double indicator technique. No reduction in sigma d was observed until microvascular pressure exceeded 70 Torr. The average sigma d for the 11 LLL in which the peak microvascular pressure was less than 70 Torr was 0.74 +/- 0.03 (SE). Above this level sigma d fell linearly with increasing Pv, with a value of 0.26 being observed after the highest Pv transient. These results suggest that protein permeability may increase in patients with NPE who develop very large increases in pulmonary vascular pressures but may not be a universal occurrence in this disorder.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1152/jappl.1989.67.1.305" target="_blank" rel="noreferrer noopener">10.1152/jappl.1989.67.1.305</a>
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
*Blood Pressure
1989
Animals
Blood Proteins/*metabolism
Dogs
Female
Hypertension/physiopathology
In Vitro Techniques
Journal of applied physiology (Bethesda, Md. : 1985)
Lung/metabolism/*physiopathology
Male
Maron M B
Pulmonary Edema/*etiology/metabolism/physiopathology
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1152/ajpheart.1992.262.1.H303" target="_blank" rel="noreferrer noopener">http://doi.org/10.1152/ajpheart.1992.262.1.H303</a>
Pages
H303–307
Issue
1
Volume
262
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Onset of exercise shifts operating point of arterial baroreflex to higher pressures.
Publisher
An entity responsible for making the resource available
The American journal of physiology
Date
A point or period of time associated with an event in the lifecycle of the resource
1992
1992-01
Subject
The topic of the resource
*Blood Pressure; *Physical Exertion; Animals; Arteries/*physiology; Denervation; Heart Rate; Kidney/physiology; Pressoreceptors/*physiology; Rabbits; Reflex/*physiology; Sinus of Valsalva/physiology; Sympathetic Nervous System/physiology
Creator
An entity primarily responsible for making the resource
DiCarlo S E; Bishop V S
Description
An account of the resource
This study was designed to test the hypothesis that the increase in sympathetic nerve activity (SNA) and mean arterial pressure (MAP) at the onset of exercise is dependent on a rapid upward shift of the operating point of the arterial baroreflex. To test this hypothesis, we recorded renal sympathetic nerve activity (RSNA) in 16 New Zealand White rabbits during treadmill running (12.6 m/min, 20% grade) under control conditions and during concomitant intravenous infusions of nitroglycerin (NTG) to attenuate the exercise pressor response. In the control condition, MAP increased 18 +/- 2 mmHg. This was associated with an increase in heart rate (HR) (104 +/- 4 beats/min) and RSNA (414 +/- 20%). The increases in RSNA (848 +/- 32%) and HR (155 +/- 5 beats/min) at the onset of exercise were significantly augmented when the rate of development of the exercise pressor response (0.3 +/- 0.03 to 0.12 +/- 0.01 mmHg/s) and the magnitudes of the pressor response (91 +/- 2 to 79 +/- 1 mmHg) were attenuated by infusions of NTG. These data suggest that at the onset of exercise the operating point of the arterial baroreflex is reset toward higher pressures. The MAP, RSNA, and HR responses to exercise were also determined in eight sinoaortic-denervated (SAD) rabbits. In the absence of a functional baroreflex, MAP (-46 +/- 2 mmHg), RSNA (-19 +/- 1%), and HR (-62 +/- 3 beats/min) decreased at the onset of exercise and recovered 1 min to -42 +/- 2, +13 +/- 1, and +9 +/- 1% of control, respectively.(ABSTRACT TRUNCATED AT 250 WORDS)
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1152/ajpheart.1992.262.1.H303" target="_blank" rel="noreferrer noopener">10.1152/ajpheart.1992.262.1.H303</a>
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
*Blood Pressure
*Physical Exertion
1992
Animals
Arteries/*physiology
Bishop V S
Denervation
DiCarlo S E
Heart Rate
Kidney/physiology
Pressoreceptors/*physiology
Rabbits
Reflex/*physiology
Sinus of Valsalva/physiology
Sympathetic Nervous System/physiology
The American journal of physiology
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1152/ajpendo.1989.256.3.E345" target="_blank" rel="noreferrer noopener">http://doi.org/10.1152/ajpendo.1989.256.3.E345</a>
Pages
E345–351
Issue
3
Volume
256
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Oxygen consumption after massive sympathetic nervous system discharge.
Publisher
An entity responsible for making the resource available
The American journal of physiology
Date
A point or period of time associated with an event in the lifecycle of the resource
1989
1989-03
Subject
The topic of the resource
*Blood Pressure; *Oxygen Consumption/drug effects; Adrenal Glands/physiology; Animals; Cardiac Output/drug effects; Dogs; Epinephrine/blood; Female; Heart Rate/drug effects; Male; Norepinephrine/blood; Pulmonary Artery/drug effects/physiology/physiopathology; Pulmonary Edema/physiopathology; Reference Values; Sympathetic Nervous System/*physiology/physiopathology; Veratrine/pharmacology
Creator
An entity primarily responsible for making the resource
Lang S A; Maron M B; Signs S A
Description
An account of the resource
We evaluated the possibility that massive, sympathetic nervous system (SNS) activation [as may precede the development of neurogenic pulmonary edema (NPE)] increases O2 demand. O2 consumption (VO2) and plasma concentrations of the calorigenic agents, epinephrine (EPI) and norepinephrine (NE) were measured in alpha-chloralose-anesthetized dogs under control conditions and for 3 h after the administration of either 1) intracisternal (ic) veratrine to activate the SNS, 2) intravenous (iv) veratrine, 3) ic saline, or 4) ic veratrine, after clamping the adrenal blood vessels. VO2 increased 31.7 +/- 3.6% (SE), and EPI and NE increased to, respectively, 30,853 +/- 8,347 and 8,176 +/- 2,104 pg/ml in the ic veratrine group. No increases in VO2 and EPI and attenuated increases in NE were observed in the ic veratrine animals with clamped adrenals. No significant increases in VO2 or catecholamine concentrations were observed after ic saline or iv veratrine administration. These data suggest that the elevated VO2 may have been mediated by adrenal catecholamines and that an increased metabolic rate may complicate the ability of patients with severe NPE to balance O2 supply with demand.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1152/ajpendo.1989.256.3.E345" target="_blank" rel="noreferrer noopener">10.1152/ajpendo.1989.256.3.E345</a>
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
*Blood Pressure
*Oxygen Consumption/drug effects
1989
Adrenal Glands/physiology
Animals
Cardiac Output/drug effects
Dogs
Epinephrine/blood
Female
Heart Rate/drug effects
Lang S A
Male
Maron M B
Norepinephrine/blood
Pulmonary Artery/drug effects/physiology/physiopathology
Pulmonary Edema/physiopathology
Reference Values
Signs S A
Sympathetic Nervous System/*physiology/physiopathology
The American journal of physiology
Veratrine/pharmacology