Browse Items · NEOMED Bibliography Database

Aspirin attenuation of alcohol-induced flushing and intoxication in Oriental and Occidental subjects.

Aspirin (ASA) was tested in a group of 8 Oriental and 3 Occidental subjects who were shown in a previous study to respond to small doses of ethanol (0.06-0.25 g/kg) with facial flushing. They were compared to a similar group of 11 non-flushing…

Tags: *Alcoholic Intoxication, 1987, Alcohol and alcoholism (Oxford, Oxfordshire). Supplement, Asian Continental Ancestry Group, Aspirin/*pharmacology, Blood Pressure/drug effects, Ethanol/*antagonists & inhibitors/pharmacology, European Continental Ancestry Group, Flushing/*chemically induced, Gaynor C R, Heart Rate/drug effects, Humans, Mehl D L, Skin Temperature/drug effects, Truitt E B Jr

Methamphetamine-gonadal steroid hormonal interactions: effects upon acute toxicity and striatal dopamine concentrations.

Methamphetamine (MA)-related deaths and nigrostriatal dopaminergic (NSDA) neurotoxicity are greater in males. The exact basis for this gender difference is not known, but data, which show that estrogen (E) can function as a protectant of both the…

Acute exercise attenuates cardiac autonomic regulation in hypertensive rats.

Dynamic exercise may be used as a safe, therapeutic approach to reduce sympathetic nerve activity at rest and thus may be beneficial for individuals with hypertension. Therefore, we tested the hypothesis that a single bout of mild to moderate dynamic…

Hemodynamic basis for cocaine-induced pulmonary edema in dogs.

We tested the hypothesis that cocaine-induced impairment of left ventricular function results in cardiogenic pulmonary edema. Mongrel dogs, anesthetized with alpha-chloralose, were injected with two doses of cocaine (5 mg/kg iv) 27 min apart. Cocaine…

Tags: 1991, Animals, Blood Pressure/drug effects, Cardiac Output/drug effects, Cocaine/*toxicity, Coronary Circulation/drug effects, Dogs, Female, Heart Rate/drug effects, Hemodynamics/*drug effects, Journal of applied physiology (Bethesda, Md. : 1985), Lang S A, Male, Maron M B, Pulmonary Circulation/drug effects, Pulmonary Edema/chemically induced/*physiopathology, Stroke Volume/drug effects, Vascular Resistance/drug effects

Acute exercise enhances nitric oxide modulation of vascular response to phenylephrine.

The influence of the release of endothelium-derived nitric oxide (NO) on the vasoconstrictor response to phenylephrine (PE) was evaluated before and after a single bout of dynamic exercise. Each rat ran on a motor-driven treadmill at

Attenuation of postexertional hypotension by cardiac afferent blockade.

Naloxone eliminates postexertional hypotension (PEH) in human and animal models. The effect of naloxone on sympathetic activity during hemorrhage and generation of arterial baroreflex function curves can be stimulated by blockade of cardiac afferent…

Tags: *Heart Arrest, *Physical Exertion, 1993, Afferent Pathways/physiology, Animals, Blood Pressure/drug effects, Collins H L, DiCarlo S E, Efferent Pathways/physiology, Heart Rate/drug effects, Hypotension/*physiopathology, Inbred SHR, Induced, Pressoreceptors/drug effects, Procainamide/pharmacology, Rats, Reference Values, The American journal of physiology

Factors involved in left ventricular dysfunction after massive sympathetic activation.

We sought to determine whether catecholamines are responsible for the depressed left ventricular (LV) function that follows massive sympathetic nervous system (SNS) activation and whether the additional myocardial energy demands of

Tags: *Ventricular Function, 1992, Animals, Blood Pressure/drug effects/physiology, Bosso F J, Catecholamines/physiology, Female, Heart Conduction System/*physiology, Heart Diseases/*physiopathology, Heart Rate/drug effects, In Vitro Techniques, Left, Male, Maron M B, Pilati C F, Rabbits, Sympathetic Nervous System/drug effects/*physiology, The American journal of physiology, Veratrine/pharmacology

Oxygen consumption after massive sympathetic nervous system discharge.

We evaluated the possibility that massive, sympathetic nervous system (SNS) activation [as may precede the development of neurogenic pulmonary edema (NPE)] increases O2 demand. O2 consumption (VO2) and plasma concentrations of the calorigenic agents,…

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