1
40
7
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.2174/156800912803987896" target="_blank" rel="noreferrer noopener">http://doi.org/10.2174/156800912803987896</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
1191-1232
Issue
9
Volume
12
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Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Dietary Phytochemicals In The Chemoprevention And Treatment Of Hepatocellular Carcinoma: In Vivo Evidence, Molecular Targets, And Clinical Relevance
Publisher
An entity responsible for making the resource available
Current Cancer Drug Targets
Date
A point or period of time associated with an event in the lifecycle of the resource
2012
2012-11
Subject
The topic of the resource
acid phenethyl ester; altered hepatic foci; black tea; carcinoma; Chemoprevention; dietary; diethylnitrosamine-induced hepatocarcinogenesis; hepatocarcinogenesis; hepatocellular; implanted; liver cancer; liver preneoplastic foci; model; multiorgan carcinogenesis model; nitrosodiethylamine-induced; nude-mice; Oncology; phenobarbital-induced hepatocarcinogenesis; phytochemicals; polyphenols; resistant hepatocyte; treatment
Creator
An entity primarily responsible for making the resource
Bishayee A; Thoppil R J; Waghray A; Kruse J A; Novotny N A; Darvesh A S
Description
An account of the resource
Hepatocellular carcinoma (HCC), one of the most common and lethal cancers, is a growing menace in modern society. Until recently, the majority of detected cases of liver cancer have been found in the developing nations of Asia and Africa; however, its occurrence has significantly increased in the United States. HCC occurs due to several etiologies, such as alcoholism, dietary carcinogens, iron overload, viral hepatitis, as well as several hepatic chronic diseases. In view of the limited treatment options, such as surgery and transplantation, a critical need exists to examine alternative approaches. The use of phytochemicals obtained from dietary sources provides a novel and fascinating preventive and therapeutic approach against HCC. Dietary phytochemicals possess potent antioxidant and anti-inflammatory properties which are extremely critical to combat the significant oxidative stress and inflammation implicated in liver cancer. An impressive number of phytochemicals have shown considerable promise as candidates for the prevention and treatment of HCC. In this article, we systematically review the in vivo pre-clinical evidence documenting the chemopreventive and therapeutic potential of several important dietary phytochemicals in HCC. This review critically examines the molecular mechanisms of the pharmacological effects of the aforementioned animal studies. Clinical and epidemiological studies are also highlighted in this review. Emerging issues such as bioavailability, dose optimization, targeted drug delivery, role of botanical extracts and synergy are also discussed. Finally, current challenges, limitations, future directions, innovative concepts and novel hypotheses for the use of dietary phytochemicals in the chemoprevention and amelioration of human HCC are presented.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.2174/156800912803987896" target="_blank" rel="noreferrer noopener">10.2174/156800912803987896</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2012
acid phenethyl ester
altered hepatic foci
Bishayee A
black tea
Carcinoma
Chemoprevention
Current cancer drug targets
Darvesh A S
Department of Pharmaceutical Sciences
Dietary
diethylnitrosamine-induced hepatocarcinogenesis
Hepatocarcinogenesis
Hepatocellular
Implanted
Journal Article or Conference Abstract Publication
Kruse J A
Liver cancer
liver preneoplastic foci
model
multiorgan carcinogenesis model
NEOMED College of Pharmacy
nitrosodiethylamine-induced
Novotny N A
nude-mice
oncology
phenobarbital-induced hepatocarcinogenesis
phytochemicals
Polyphenols
resistant hepatocyte
Thoppil R J
Treatment
Waghray A
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1002/mc.21860" target="_blank" rel="noreferrer noopener">http://doi.org/10.1002/mc.21860</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
304-317
Issue
4
Volume
52
Search for Full-text
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Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Black Currant Phytoconstituents Exert Chemoprevention Of Diethylnitrosamine-initiated Hepatocarcinogenesis By Suppression Of The Inflammatory Response
Publisher
An entity responsible for making the resource available
Molecular Carcinogenesis
Date
A point or period of time associated with an event in the lifecycle of the resource
2013
2013-04
Subject
The topic of the resource
Biochemistry & Molecular Biology; cancer-cell proliferation; Chemoprevention; cyclooxygenase-2; cyclooxygenase-2 inhibitors; gamma-glutamyl-transferase; heat-shock proteins; heat-shock proteins; hepatocarcinogenesis; human hepatocellular-carcinoma; Inflammation; liver cancer; molecular chaperones; nf-kappa-b; nuclear factor-B; Oncology; ribes-nigrum; signaling; united-states
Creator
An entity primarily responsible for making the resource
Bishayee A; Thoppil R J; Mandal A; Darvesh A S; Ohanyan V; Meszaros J G; Haznagy-Radnai E; Hohmann J; Bhatia D
Description
An account of the resource
Black currant fruits containing high amounts of anthocyanins are known to possess potent antioxidant and anti-inflammatory properties. We have previously reported that anthocyanin-rich black currant skin extract (BCSE) inhibits diethylnitrosamine (DENA)-initiated hepatocarcinogenesis in rats although the underlying mechanisms are not fully understood. Our present study investigates the anti-inflammatory mechanisms of BCSE during DENA rat liver carcinogenesis. Dietary BCSE (100 or 500mg/kg) treatment for 22wk afforded a striking inhibition of DENA-induced hepatic gamma-glutamyl transpeptidase-positive preneoplastic foci in a dose-responsive fashion. There was a significant increase in hepatic expression of heat shock proteins (HSP70 and HSP90), cyclooxygenase-2, and nuclear factor-B (NF-B) in DENA-exposed rat livers. Dietary BCSE dose-dependently abrogated all these elevated inflammatory markers. The possible cardiotoxicity of BCSE was assessed by monitoring cardiac functions using transthoracic echocardiography. BCSE-mediated anti-inflammatory effects during rat liver carcinogenesis have been achieved without any cardiotoxicity. Our results provide convincing evidence, for the very first time, that suppression of the inflammatory cascade through modulation of the NF-B signaling pathway could be implicated, at least in part, in the chemopreventive effects of black currant bioactive phytoconstituents against experimental hepatocarcinogenesis. These results coupled with an excellent safety profile of BCSE support the development of black currant phytochemicals for the chemoprevention of inflammation-driven hepatocellular cancer. (c) 2011 Wiley Periodicals, Inc.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1002/mc.21860" target="_blank" rel="noreferrer noopener">10.1002/mc.21860</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2013
Bhatia D
Biochemistry & Molecular Biology
Bishayee A
cancer-cell proliferation
Chemoprevention
cyclooxygenase-2
cyclooxygenase-2 inhibitors
Darvesh A S
Department of Pharmaceutical Sciences
gamma-glutamyl-transferase
Háznagy-Radnai E
Heat-Shock Proteins
Hepatocarcinogenesis
Hohmann J
human hepatocellular-carcinoma
Inflammation
Journal Article or Conference Abstract Publication
Liver cancer
Mandal A
Meszaros J G
Molecular carcinogenesis
molecular chaperones
NEOMED College of Pharmacy
nf-kappa-b
nuclear factor-B
Ohanyan V
oncology
ribes-nigrum
Signaling
Thoppil R J
united-states
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1016/j.jnutbio.2012.04.009" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/j.jnutbio.2012.04.009</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
178-187
Issue
1
Volume
24
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
<p>Users with a NEOMED Library login can search for full-text journal articles at the following url: <a href="https://libraryguides.neomed.edu/home">https://libraryguides.neomed.edu/home</a></p>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Pomegranate Phytoconstituents Blunt The Inflammatory Cascade In A Chemically Induced Rodent Model Of Hepatocellular Carcinogenesis
Publisher
An entity responsible for making the resource available
Journal of Nutritional Biochemistry
Date
A point or period of time associated with an event in the lifecycle of the resource
2013
2013-01
Subject
The topic of the resource
Biochemistry & Molecular Biology; cancer chemoprevention; Chemoprevention; cyclooxygenase-2; cyclooxygenase-2; factor-kappa-b; gene-expression; heat-shock proteins; hepatocarcinogenesis; human-disease; Inflammation; inhibitors; molecular chaperones; nitric-oxide synthase; Nuclear factor-kappaB; Nutrition & Dietetics; oxidative stress; Pomegranate; punica-granatum l.
Creator
An entity primarily responsible for making the resource
Bishayee A; Thoppil R J; Darvesh A S; Ohanyan V; Meszaros J G; Bhatia D
Description
An account of the resource
Liver cancer, predominantly hepatocellular carcinoma (HCC), represents a complex and fatal malignancy driven primarily by oxidative stress and inflammation. Due to dismal prognosis and limited therapeutic intervention, chemoprevention has emerged as a viable approach to reduce the morbidity and mortality of HCC. Pomegranate fruit is a rich source of phytochemicals endowed with potent antioxidant and anti-inflammatory properties. We previously reported that pomegranate phytochemicals inhibit diethylnitrosamine (DENA)-initiated hepatocarcinogenesis in rats though nuclear factor E2-related factor 2 (Nrf2)-mediated antioxidant mechanisms. Since Nrf2 also acts as a key mediator of the nuclear factor-kappaB (NF-kappa B)-regulated inflammatory pathway, our present study investigated the anti-inflammatory mechanisms of a pomegranate emulsion (PE) during DENA-induced rat hepatocarcinogenesis. Rats were administered with PE (1 or 10 g/kg) 4 weeks before and 18 weeks following DENA initiation. There was a significant increase in hepatic expressions of inducible nitric oxide synthase, 3-nitrotyrosine, heat shock protein 70 and 90, cyclooxygenase-2 and NF-kappa B in DENA-exposed rat livers. PE dose-dependently suppressed all aforementioned elevated inflammatory markers. A conspicuous finding of this study involves lack of cardiotoxicity of PE as assessed by monitoring cardiac function using noninvasive echocardiography. Our results provide substantial evidence that suppression of the inflammatory cascade through modulation of NF-kappa B signaling pathway may represent a novel mechanism of liver tumor inhibitory effects of PE against experimental hepatocarcinogenesis. Data presented here coupled with those of our earlier study underline the importance of simultaneously targeting two interconnected molecular circuits, namely, Nrf2-mediated redox signaling and NF-kappa B-regulated inflammatory pathway, by pomegranate phytoconstituents to achieve chemoprevention of HCC. (C) 2013 Elsevier Inc. All rights reserved.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1016/j.jnutbio.2012.04.009" target="_blank" rel="noreferrer noopener">10.1016/j.jnutbio.2012.04.009</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2013
Bhatia D
Biochemistry & Molecular Biology
Bishayee A
cancer chemoprevention
Chemoprevention
cyclooxygenase-2
Darvesh A S
Department of Pharmaceutical Sciences
factor-kappa-b
gene-expression
Heat-Shock Proteins
Hepatocarcinogenesis
human-disease
Inflammation
inhibitors
Journal Article or Conference Abstract Publication
Journal of Nutritional Biochemistry
Meszaros J G
molecular chaperones
NEOMED College of Pharmacy
nitric-oxide synthase
Nuclear factor-kappaB
Nutrition & Dietetics
Ohanyan V
Oxidative Stress
Pomegranate
punica-granatum l.
Thoppil R J
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.2174/156800912803987896" target="_blank" rel="noreferrer noopener">http://doi.org/10.2174/156800912803987896</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
1191-1232
Issue
9
Volume
12
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
<p>Users with a NEOMED Library login can search for full-text journal articles at the following url: <a href="https://libraryguides.neomed.edu/home">https://libraryguides.neomed.edu/home</a></p>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Dietary Phytochemicals In The Chemoprevention And Treatment Of Hepatocellular Carcinoma: In Vivo Evidence, Molecular Targets, And Clinical Relevance
Publisher
An entity responsible for making the resource available
Current Cancer Drug Targets
Date
A point or period of time associated with an event in the lifecycle of the resource
2012
2012-11
Subject
The topic of the resource
acid phenethyl ester; altered hepatic foci; black tea; carcinoma; chemoprevention; dietary; diethylnitrosamine-induced hepatocarcinogenesis; hepatocarcinogenesis; hepatocellular; implanted; liver cancer; liver preneoplastic foci; model; multiorgan carcinogenesis model; nitrosodiethylamine-induced; nude-mice; Oncology; phenobarbital-induced hepatocarcinogenesis; phytochemicals; polyphenols; resistant hepatocyte; treatment
Creator
An entity primarily responsible for making the resource
Bishayee A; Thoppil R J; Waghray A; Kruse J A; Novotny N A; Darvesh A S
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.2174/156800912803987896" target="_blank" rel="noreferrer noopener">10.2174/156800912803987896</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2012
acid phenethyl ester
altered hepatic foci
Bishayee A
black tea
Carcinoma
Chemoprevention
Current cancer drug targets
Darvesh A S
Department of Pharmaceutical Sciences
Dietary
diethylnitrosamine-induced hepatocarcinogenesis
Hepatocarcinogenesis
Hepatocellular
Implanted
Kruse J A
Liver cancer
liver preneoplastic foci
model
multiorgan carcinogenesis model
NEOMED College of Pharmacy
nitrosodiethylamine-induced
Novotny N A
nude-mice
oncology
phenobarbital-induced hepatocarcinogenesis
phytochemicals
Polyphenols
resistant hepatocyte
Thoppil R J
Treatment
Waghray A
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1002/mc.21860" target="_blank" rel="noreferrer noopener">http://doi.org/10.1002/mc.21860</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
304-317
Issue
4
Volume
52
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
<p>Users with a NEOMED Library login can search for full-text journal articles at the following url: <a href="https://libraryguides.neomed.edu/home">https://libraryguides.neomed.edu/home</a></p>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Black Currant Phytoconstituents Exert Chemoprevention Of Diethylnitrosamine-initiated Hepatocarcinogenesis By Suppression Of The Inflammatory Response
Publisher
An entity responsible for making the resource available
Molecular Carcinogenesis
Date
A point or period of time associated with an event in the lifecycle of the resource
2013
2013-04
Subject
The topic of the resource
Biochemistry & Molecular Biology; cancer-cell proliferation; chemoprevention; cyclooxygenase-2; cyclooxygenase-2 inhibitors; gamma-glutamyl-transferase; heat-shock proteins; heat-shock proteins; hepatocarcinogenesis; human hepatocellular-carcinoma; inflammation; liver cancer; molecular chaperones; nf-kappa-b; nuclear factor-B; Oncology; ribes-nigrum; signaling; united-states
Creator
An entity primarily responsible for making the resource
Bishayee A; Thoppil R J; Mandal A; Darvesh A S; Ohanyan V; Meszaros J G; Haznagy-Radnai E; Hohmann J; Bhatia D
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1002/mc.21860" target="_blank" rel="noreferrer noopener">10.1002/mc.21860</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2013
Bhatia D
Biochemistry & Molecular Biology
Bishayee A
cancer-cell proliferation
Chemoprevention
cyclooxygenase-2
cyclooxygenase-2 inhibitors
Darvesh A S
Department of Pharmaceutical Sciences
gamma-glutamyl-transferase
Háznagy-Radnai E
Heat-Shock Proteins
Hepatocarcinogenesis
Hohmann J
human hepatocellular-carcinoma
Inflammation
Liver cancer
Mandal A
Meszaros J G
Molecular carcinogenesis
molecular chaperones
NEOMED College of Pharmacy
nf-kappa-b
nuclear factor-B
Ohanyan V
oncology
ribes-nigrum
Signaling
Thoppil R J
united-states
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1016/j.jnutbio.2012.04.009" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/j.jnutbio.2012.04.009</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
178-187
Issue
1
Volume
24
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
<p>Users with a NEOMED Library login can search for full-text journal articles at the following url: <a href="https://libraryguides.neomed.edu/home">https://libraryguides.neomed.edu/home</a></p>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Pomegranate Phytoconstituents Blunt The Inflammatory Cascade In A Chemically Induced Rodent Model Of Hepatocellular Carcinogenesis
Publisher
An entity responsible for making the resource available
Journal of Nutritional Biochemistry
Date
A point or period of time associated with an event in the lifecycle of the resource
2013
2013-01
Subject
The topic of the resource
Biochemistry & Molecular Biology; cancer chemoprevention; chemoprevention; cyclooxygenase-2; cyclooxygenase-2; factor-kappa-b; gene-expression; heat-shock proteins; hepatocarcinogenesis; human-disease; inflammation; inhibitors; molecular chaperones; nitric-oxide synthase; Nuclear factor-kappaB; Nutrition & Dietetics; oxidative stress; Pomegranate; punica-granatum l.
Creator
An entity primarily responsible for making the resource
Bishayee A; Thoppil R J; Darvesh A S; Ohanyan V; Meszaros J G; Bhatia D
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1016/j.jnutbio.2012.04.009" target="_blank" rel="noreferrer noopener">10.1016/j.jnutbio.2012.04.009</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2013
Bhatia D
Biochemistry & Molecular Biology
Bishayee A
cancer chemoprevention
Chemoprevention
cyclooxygenase-2
Darvesh A S
Department of Pharmaceutical Sciences
factor-kappa-b
gene-expression
Heat-Shock Proteins
Hepatocarcinogenesis
human-disease
Inflammation
inhibitors
Journal of Nutritional Biochemistry
Meszaros J G
molecular chaperones
NEOMED College of Pharmacy
nitric-oxide synthase
Nuclear factor-kappaB
Nutrition & Dietetics
Ohanyan V
Oxidative Stress
Pomegranate
punica-granatum l.
Thoppil R J
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.4254/wjh.v3.i9.228" target="_blank" rel="noreferrer noopener">http://doi.org/10.4254/wjh.v3.i9.228</a>
Pages
228–249
Issue
9
Volume
3
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Terpenoids as potential chemopreventive and therapeutic agents in liver cancer.
Publisher
An entity responsible for making the resource available
World journal of hepatology
Date
A point or period of time associated with an event in the lifecycle of the resource
2011
2011-09
Subject
The topic of the resource
Apoptosis; Chemoprevention; Treatment; Cell cycle; Hepatocarcinogenesis; Hepatocellular carcinoma; Liver cancer cells; Terpenoids
Creator
An entity primarily responsible for making the resource
Thoppil Roslin J; Bishayee Anupam
Description
An account of the resource
Despite significant advances in medicine, liver cancer, predominantly hepatocellular carcinoma remains a major cause of death in the United States as well as the rest of the world. As limited treatment options are currently available to patients with liver cancer, novel preventive control and effective therapeutic approaches are considered to be reasonable and decisive measures to combat this disease. Several naturally occurring dietary and non-dietary phytochemicals have shown enormous potential in the prevention and treatment of several cancers, especially those of the gastrointestinal tract. Terpenoids, the largest group of phytochemicals, traditionally used for medicinal purposes in India and China, are currently being explored as anticancer agents in clinical trials. Terpenoids (also called "isoprenoids") are secondary metabolites occurring in most organisms, particularly plants. More than 40 000 individual terpenoids are known to exist in nature with new compounds being discovered every year. A large number of terpenoids exhibit cytotoxicity against a variety of tumor cells and cancer preventive as well as anticancer efficacy in preclinical animal models. This review critically examines the potential role of naturally occurring terpenoids, from diverse origins, in the chemoprevention and treatment of liver tumors. Both in vitro and in vivo effects of these agents and related cellular and molecular mechanisms are highlighted. Potential challenges and future directions involved in the advancement of these promising natural compounds in the chemoprevention and therapy of human liver cancer are also discussed.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.4254/wjh.v3.i9.228" target="_blank" rel="noreferrer noopener">10.4254/wjh.v3.i9.228</a>
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
2011
Apoptosis
Bishayee Anupam
Cell cycle
Chemoprevention
Hepatocarcinogenesis
Hepatocellular carcinoma
Liver cancer cells
Terpenoids
Thoppil Roslin J
Treatment
World journal of hepatology