1
40
8
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1016/0028-3932(89)90088-2" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/0028-3932(89)90088-2</a>
Pages
31–39
Issue
1
Volume
27
Dublin Core
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Title
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The development of long-term potentiation in hippocampus and neocortex.
Publisher
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Neuropsychologia
Date
A point or period of time associated with an event in the lifecycle of the resource
1989
1989
Subject
The topic of the resource
Female; Animals; Neurons/physiology; Rats; Electric Stimulation; *Synaptic Transmission; Aging/*physiology; Synapses/physiology; Hippocampus/*physiology; Cerebral Cortex/*physiology; Memory/*physiology; Evoked Potentials; Mental Recall/*physiology; Visual Cortex/physiology; Visual Pathways/physiology; Inbred Strains
Creator
An entity primarily responsible for making the resource
Teyler T J; Perkins A T 4th; Harris K M
Description
An account of the resource
The development of long-term potentiation (LTP), an enduring alteration in synaptic efficacy following afferent activation, was examined in CA1 hippocampus and primary visual cortex of rat. Both regions show little LTP prior to postnatal day 5, demonstrate a maximal potentiated response around postnatal day 15, and a subsequent decline to adult levels. These results are discussed with respect to the underlying mechanism of action and behavioral significance of these critical-period phenomena.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1016/0028-3932(89)90088-2" target="_blank" rel="noreferrer noopener">10.1016/0028-3932(89)90088-2</a>
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
*Synaptic Transmission
1989
Aging/*physiology
Animals
Cerebral Cortex/*physiology
Electric Stimulation
Evoked Potentials
Female
Harris K M
Hippocampus/*physiology
Inbred Strains
Memory/*physiology
Mental Recall/*physiology
Neurons/physiology
Neuropsychologia
Perkins A T 4th
Rats
Synapses/physiology
Teyler T J
Visual Cortex/physiology
Visual Pathways/physiology
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1016/0165-0270(89)90015-0" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/0165-0270(89)90015-0</a>
Pages
101–108
Issue
1
Volume
28
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Comparative aspects of hippocampal and neocortical long-term potentiation.
Publisher
An entity responsible for making the resource available
Journal of neuroscience methods
Date
A point or period of time associated with an event in the lifecycle of the resource
1989
1989-05
Subject
The topic of the resource
Animals; Electric Stimulation; Action Potentials; Hippocampus/*physiology; Visual Cortex/*physiology; Evoked Potentials; Memory/physiology; Receptors; N-Methyl-D-Aspartate; Neurotransmitter/*physiology
Creator
An entity primarily responsible for making the resource
Teyler T J
Description
An account of the resource
Long-term potentiation (LTP) is a candidate for the synaptic alternations underlying memory storage in the mammalian CNS. In this chapter LTP in hippocampus and in visual neocortex are compared. Comparisons of the optimal tetanus parameters revealed that 2-3 trains of high-frequency stimulation (100-400 Hz) delivered within a brief period of time (minutes) results in maximal potentiation in hippocampal synapses. In contrast, the parameters most effective in neocortex were either low-frequency (2 Hz for 60 min) or high-frequency bursts (100 Hz, 100 ms train at 1/5 s for 10 min), both of which deliver at least an order of magnitude more afferent activation than that required for hippocampus. Hippocampal population spike potentiation averages 250% and the population excitatory postsynaptic potential (EPSP) potentiation averages 50%. Neocortical LTP also averages about 50%. The expression of LTP requires about 5 min in CA1 hippocampus, whereas about 30 min are required for expression of neocortical potentiation. Both hippocampus and visual neocortex display an enhanced potentiation early in development, with a later stabilization at lower adult levels. Centering at postnatal day 15, hippocampal CA1 displays an LTP magnitude that is over twice that seen at day 60. Neocortical responses display a similar peak at postnatal day 15 and a subsequent adult stabilization at approximately half of the day 15 maximum. Both tissues first display LTP during the early stages of synapse formation between postnatal days 6-10. The role of the NMDA receptor is implicated in aspects of both hippocampal and neocortical LTP.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1016/0165-0270(89)90015-0" target="_blank" rel="noreferrer noopener">10.1016/0165-0270(89)90015-0</a>
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
1989
Action Potentials
Animals
Electric Stimulation
Evoked Potentials
Hippocampus/*physiology
Journal of neuroscience methods
Memory/physiology
N-Methyl-D-Aspartate
Neurotransmitter/*physiology
Receptors
Teyler T J
Visual Cortex/*physiology
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1152/jn.1993.69.5.1541" target="_blank" rel="noreferrer noopener">http://doi.org/10.1152/jn.1993.69.5.1541</a>
Pages
1541–1555
Issue
5
Volume
69
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Role of HCO3- ions in depolarizing GABAA receptor-mediated responses in pyramidal cells of rat hippocampus.
Publisher
An entity responsible for making the resource available
Journal of neurophysiology
Date
A point or period of time associated with an event in the lifecycle of the resource
1993
1993-05
Subject
The topic of the resource
Afferent Pathways/physiology; Animals; Bicarbonates/*metabolism; Culture Techniques; Electric Stimulation; GABA-A/*physiology; gamma-Aminobutyric Acid/physiology; Hippocampus/*physiology; Interneurons/physiology; Membrane Potentials/physiology; Neurons/physiology; Rats; Receptors; Synapses/physiology; Synaptic Transmission/*physiology
Creator
An entity primarily responsible for making the resource
Grover L M; Lambert N A; Schwartzkroin P A; Teyler T J
Description
An account of the resource
1. Activation of GABAA receptors can produce both hyperpolarizing and depolarizing responses in CA1 pyramidal cells. The hyperpolarizing response is mediated by a Cl- conductance, but the ionic basis of the depolarizing response is not clear. We compared the GABAA receptor-mediated depolarizations induced by synaptically released gamma-aminobutyric acid [GABA; depolarizing inhibitory postsynaptic potentials (dIPSPs)] with those produced by exogenous GABA (depolarizing GABA responses). Short trains of high-frequency (200 Hz) stimuli were used to generate dIPSPs. We found that dIPSPs generated by trains of stimuli and depolarizing responses to exogenous GABA were accompanied by a conductance increase and had a similar reversal potential, indicating a similar ionic basis for both responses. 2. We wished to determine whether an HCO3- current contributed to the GABAA-mediated depolarizations. We found that dIPSPs and depolarizing GABA responses were sensitive to perfusion with HCO3(-)-free medium. Interpretation of these data was complicated by the mixed nature of the responses: dIPSPs were invariably accompanied by conventional, Cl(-)-mediated fast hyperpolarizing IPSPs (fIPSPs), and response to exogenous GABA usually consisted of biphasic hyperpolarizing and depolarizing responses. However, it was sometimes possible to elicit responses to GABA that appeared purely depolarizing (monophasic depolarizing GABA responses). 3. We analyzed monophasic depolarizing GABA responses and found no change in reversal potential when slices were perfused with HCO(3-)-free medium. We also made whole-cell recordings from CA1 pyramidal cells, attempting to reduce [HCO3-]i, and compared the reversal potential for monophasic depolarizing GABA responses with similar responses recorded with fine intracellular microelectrodes. We found no difference in reversal potential. We also examined effects of the carbonic anhydrase inhibitor acetazolamide (ACTZ) on depolarizing GABA responses. ACTZ reduced these responses but did not change their reversal potential. 4. Effects of HCO(3-)-free medium were not specific to GABAA receptor-mediated responses. GABAB receptor-mediated slow IPSPs (sIPSPs) were also reduced, as were excitatory postsynaptic potentials (EPSPs). Analyses of field potentials and spontaneous fIPSPs suggested a decrease in presynaptic excitability during perfusion with HCO(3-)-free medium. In addition, pyramidal cells showed decreased input resistance when perfused with HCO(3-)-free medium. 5. The sensitivity of GABAA receptor-mediated depolarizations to HCO(3-)-free medium can be explained by a decrease in presynaptic excitability and an increased resting conductance in postsynaptic neurons.(ABSTRACT TRUNCATED AT 400 WORDS)
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1152/jn.1993.69.5.1541" target="_blank" rel="noreferrer noopener">10.1152/jn.1993.69.5.1541</a>
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
1993
Afferent Pathways/physiology
Animals
Bicarbonates/*metabolism
Culture Techniques
Electric Stimulation
GABA-A/*physiology
gamma-Aminobutyric Acid/physiology
Grover L M
Hippocampus/*physiology
Interneurons/physiology
Journal of neurophysiology
Lambert N A
Membrane Potentials/physiology
Neurons/physiology
Rats
Receptors
Schwartzkroin P A
Synapses/physiology
Synaptic Transmission/*physiology
Teyler T J
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1152/jn.1991.66.5.1538" target="_blank" rel="noreferrer noopener">http://doi.org/10.1152/jn.1991.66.5.1538</a>
Pages
1538–1548
Issue
5
Volume
66
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Hyperpolarizing and depolarizing GABAA receptor-mediated dendritic inhibition in area CA1 of the rat hippocampus.
Publisher
An entity responsible for making the resource available
Journal of neurophysiology
Date
A point or period of time associated with an event in the lifecycle of the resource
1991
1991-11
Subject
The topic of the resource
2-Amino-5-phosphonovalerate/pharmacology; Animals; Bicuculline/analogs & derivatives/pharmacology; Chlorides/pharmacology; Dendrites/drug effects/*physiology; Evoked Potentials/drug effects; GABA-A Receptor Antagonists; GABA-A/drug effects/*physiology; Hippocampus/*physiology; In Vitro Techniques; Kinetics; Mathematics; Membrane Potentials/drug effects; Models; Neurological; Neurons/drug effects/*physiology; Organophosphorus Compounds/pharmacology; Pyramidal Tracts/drug effects/*physiology; Quinoxalines/pharmacology; Rats; Receptors; Synapses/drug effects/physiology
Creator
An entity primarily responsible for making the resource
Lambert N A; Borroni A M; Grover L M; Teyler T J
Description
An account of the resource
1. gamma-Aminobutyric acidA (GABAA) receptor-mediated inhibition of pyramidal neuron dendrites was studied in area CA1 of the rat hippocampal slice preparation with the use of intracellular and extracellular recording and one-dimensional current source-density (CSD) analysis. 2. Electrical stimulation of Schaffer collateral/commissural fibers evoked monosynaptic excitatory postsynaptic potentials (EPSPs) and population EPSPs, which were followed by biphasic inhibitory postsynaptic potentials (IPSPs). In the presence of the excitatory amino acid receptor antagonists 6,7-dinitroquinoxaline-2,3-dione (DNQX) and D,L-2-amino-5-phosphonovalerate (APV), stimulation in stratum radiatum evoked monosynaptic fast, GABAA and late, GABAB receptor-mediated IPSPs and fast and late positive field potentials recorded in s. radiatum. 3. Fast monosynaptic IPSPs and fast positive field potentials evoked in the presence of DNQX and APV were reversibly abolished by the GABAA receptor antagonist bicuculline methiodide (BMI; 30 microM) and were not changed by the GABAB receptor antagonist
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1152/jn.1991.66.5.1538" target="_blank" rel="noreferrer noopener">10.1152/jn.1991.66.5.1538</a>
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
1991
2-Amino-5-phosphonovalerate/pharmacology
Animals
Bicuculline/analogs & derivatives/pharmacology
Borroni A M
Chlorides/pharmacology
Dendrites/drug effects/*physiology
Evoked Potentials/drug effects
GABA-A Receptor Antagonists
GABA-A/drug effects/*physiology
Grover L M
Hippocampus/*physiology
In Vitro Techniques
Journal of neurophysiology
Kinetics
Lambert N A
Mathematics
Membrane Potentials/drug effects
Models
Neurological
Neurons/drug effects/*physiology
Organophosphorus Compounds/pharmacology
Pyramidal Tracts/drug effects/*physiology
Quinoxalines/pharmacology
Rats
Receptors
Synapses/drug effects/physiology
Teyler T J
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1038/347477a0" target="_blank" rel="noreferrer noopener">http://doi.org/10.1038/347477a0</a>
Pages
477–479
Issue
6292
Volume
347
Dublin Core
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Title
A name given to the resource
Two components of long-term potentiation induced by different patterns of afferent activation.
Publisher
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Nature
Date
A point or period of time associated with an event in the lifecycle of the resource
1990
1990-10
Subject
The topic of the resource
2-Amino-5-phosphonovalerate/*pharmacology; Afferent Pathways/physiology; Animals; Calcium/*physiology; Hippocampus/*physiology; In Vitro Techniques; Membrane Potentials; Memory/*physiology; N-Methyl-D-Aspartate/*physiology; Neuronal Plasticity; Receptors; Synaptic Transmission; Time Factors
Creator
An entity primarily responsible for making the resource
Grover L M; Teyler T J
Description
An account of the resource
Long-term potentiation (LTP) of excitatory synaptic transmission could be a mechanism underlying memory. Induction of LTP requires Ca2+ influx into postsynaptic neurons through ion channels gated by NMDA (N-methyl-D-aspartate) receptors in hippocampus (area CA1 and dentate gyrus) and neocortex. Here we report that a component of LTP not requiring the activation of NMDA receptors can be induced in area CA1. The component is dependent on tetanus frequency, requires increases in postsynaptic intracellular Ca2+ concentrations, and is suppressed by an antagonist of voltage-dependent Ca2+ channels.
Identifier
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<a href="http://doi.org/10.1038/347477a0" target="_blank" rel="noreferrer noopener">10.1038/347477a0</a>
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Information about rights held in and over the resource
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
1990
2-Amino-5-phosphonovalerate/*pharmacology
Afferent Pathways/physiology
Animals
Calcium/*physiology
Grover L M
Hippocampus/*physiology
In Vitro Techniques
Membrane Potentials
Memory/*physiology
N-Methyl-D-Aspartate/*physiology
Nature
Neuronal Plasticity
Receptors
Synaptic Transmission
Teyler T J
Time Factors
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1016/0306-4522(92)90072-a" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/0306-4522(92)90072-a</a>
Pages
7–11
Issue
1
Volume
49
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
N-methyl-D-aspartate receptor-independent long-term potentiation in area CA1 of rat hippocampus: input-specific induction and preclusion in a non-tetanized pathway.
Publisher
An entity responsible for making the resource available
Neuroscience
Date
A point or period of time associated with an event in the lifecycle of the resource
1992
1992-07
Subject
The topic of the resource
2-Amino-5-phosphonovalerate/*pharmacology; Animals; Electron; Evoked Potentials/drug effects; Hippocampus/*physiology; In Vitro Techniques; Microscopy; N-Methyl-D-Aspartate/*physiology; N-Methylaspartate/*pharmacology; Pyramidal Tracts/drug effects/*physiology; Rats; Receptors; Time Factors
Creator
An entity primarily responsible for making the resource
Grover L M; Teyler T J
Description
An account of the resource
We previously reported that an N-methyl-D-aspartate receptor-independent component of long-term potentiation with an apparent delayed onset can be induced in area CA1 of the hippocampus. Here we show that some but not all of this delay in onset can be accounted for by a transient heterosynaptic depression. We also show that N-methyl-D-aspartate receptor-independent long-term potentiation is induced only in the input pathway tetanized, and not in a second pathway. However, prior induction of N-methyl-D-aspartate receptor-independent long-term potentiation in one pathway precludes later induction in an independent pathway. Calcium entry through dihydropyridine-sensitive Ca2+ channels may be a critical step for induction of N-methyl-D-aspartate receptor-independent long-term potentiation in area CA1 [Grover L. M. and Teyler T.J. (1990) Nature 347,
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1016/0306-4522(92)90072-a" target="_blank" rel="noreferrer noopener">10.1016/0306-4522(92)90072-a</a>
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
1992
2-Amino-5-phosphonovalerate/*pharmacology
Animals
Electron
Evoked Potentials/drug effects
Grover L M
Hippocampus/*physiology
In Vitro Techniques
Microscopy
N-Methyl-D-Aspartate/*physiology
N-Methylaspartate/*pharmacology
Neuroscience
Pyramidal Tracts/drug effects/*physiology
Rats
Receptors
Teyler T J
Time Factors
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1016/0165-0270(94)00188-m" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/0165-0270(94)00188-m</a>
Pages
11–17
Issue
1
Volume
59
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Synaptic plasticity in the hippocampal slice: functional consequences.
Publisher
An entity responsible for making the resource available
Journal of neuroscience methods
Date
A point or period of time associated with an event in the lifecycle of the resource
1995
1995-06
Subject
The topic of the resource
Animals; Calcium Channels/physiology; Hippocampus/*physiology; Long-Term Potentiation/physiology; Models; N-Methyl-D-Aspartate/physiology; Neurological; Neuronal Plasticity/*physiology; Receptors; Synapses/*physiology
Creator
An entity primarily responsible for making the resource
Teyler T J; Cavus I; Coussens C
Description
An account of the resource
There are 3 known forms of synaptic plasticity at CNS synapses: long-term potentiation (LTP) mediated by NMDA receptor activation, LTP mediated by voltage-dependent calcium channel (VDCC) activation, and long-term depression (LTD) mediated by the NMDA receptor. All 3 forms of synaptic plasticity can be observed in hippocampal CAl cells, all are induced by afferent activation, all involve Ca2+ influx, and all activate Ca(2+)-dependent mechanisms. We consider the functional consequences of the presence of 3, sometime opposing, forms of synaptic plasticity at the same synapse. We suggest that the 2 forms of LTP have different consequences for the synapse. We postulate that the co-existence of potentiating and depressing capabilities influences the network processing capabilities of neural networks.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1016/0165-0270(94)00188-m" target="_blank" rel="noreferrer noopener">10.1016/0165-0270(94)00188-m</a>
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
1995
Animals
Calcium Channels/physiology
Cavus I
Coussens C
Hippocampus/*physiology
Journal of neuroscience methods
Long-Term Potentiation/physiology
Models
N-Methyl-D-Aspartate/physiology
Neurological
Neuronal Plasticity/*physiology
Receptors
Synapses/*physiology
Teyler T J
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1006/nlme.2001.4016" target="_blank" rel="noreferrer noopener">http://doi.org/10.1006/nlme.2001.4016</a>
Pages
229–238
Issue
3
Volume
76
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Depotentiation of vdccLTP requires NMDAR activation.
Publisher
An entity responsible for making the resource available
Neurobiology of learning and memory
Date
A point or period of time associated with an event in the lifecycle of the resource
2001
2001-11
Subject
The topic of the resource
2-Amino-5-phosphonovalerate/pharmacology; Animals; Calcium Channel Blockers/*pharmacology; Calcium Channels/drug effects/*physiology; Electric Stimulation; Electrophysiology; Hippocampus/*physiology; Long-Evans; Long-Term Potentiation/*physiology; Male; N-Methyl-D-Aspartate/drug effects/*physiology; Nifedipine/*pharmacology; Organ Culture Techniques; Rats; Receptors; Synaptic Transmission/*physiology
Creator
An entity primarily responsible for making the resource
Morgan S L; Coussens C M; Teyler T J
Description
An account of the resource
Long-term potentiation is an enduring increase in synaptic efficacy following repeated stimulation of afferent fibers that is thought to underlie memory. In area CA1 of the hippocampus at least two forms of synaptic potentiation coexist at the same synapses; nmdaLTP and vdccLTP. NmdaLTP is induced by Ca2+ entry through NMDARs and is dependent on serine/threonine kinase activation, while vdccLTP is induced through Ca2+ entry through VDCCs and is dependent on tyrosine kinase activation. Depotentiation is a mechanism known to reverse nmdaLTP through phosphatase activation. The depotentiation of vdccLTP has not been previously investigated. We used hippocampal slices (area CA1) from male Long-Evans rats to induce vdccLTP with a 200-Hz tetanus in the presence of 50 microM APV. The 200-Hz tetanus resulted in a slowly developing vdccLTP that remained stable for at least 30 min. Thirty minutes after vdccLTP was induced, a low-frequency tetanus (3, 10, 20, 30, or 40 Hz) was applied in the presence of APV in an attempt to depotentiate vdccLTP. The 3- and 10-Hz low-frequency tetani resulted in no depotentiation. The 20- and 30-Hz tetani partially depotentiated vdccLTP (by approximately 13%), whereas the 40-Hz tetanus resulted in further potentiation. When APV was washed out prior to the 3-Hz low-frequency tetanus, the vdccLTP was completely depotentiated–presumably by NMDAR mechanisms. Our results indicate that vdccLTP is resistant to depotentiation under low-frequency stimulation conditions that readily depotentiate nmdaLTP. As tetanus frequencies are increased a small depotentiation is observed, suggesting that vdccLTP can be depotentiated to a small extent. When NMDARs are unblocked, vdccLTP can be completely depotentiated by a 3-Hz low-frequency tetanus, suggesting that vdccLTP can be depotentiated via activation of NMDAR mechanisms.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1006/nlme.2001.4016" target="_blank" rel="noreferrer noopener">10.1006/nlme.2001.4016</a>
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
2-Amino-5-phosphonovalerate/pharmacology
2001
Animals
Calcium Channel Blockers/*pharmacology
Calcium Channels/drug effects/*physiology
Coussens C M
Electric Stimulation
Electrophysiology
Hippocampus/*physiology
Long-Evans
Long-Term Potentiation/*physiology
Male
Morgan S L
N-Methyl-D-Aspartate/drug effects/*physiology
Neurobiology of learning and memory
Nifedipine/*pharmacology
Organ Culture Techniques
Rats
Receptors
Synaptic Transmission/*physiology
Teyler T J