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9
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<a href="http://doi.org/10.1096/fasebj.2020.34.s1.01787" target="_blank" rel="noreferrer noopener">http://doi.org/10.1096/fasebj.2020.34.s1.01787</a>
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1
Volume
34
ISSN
0892-6638
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September 2020 List
NEOMED College
NEOMED College of Medicine
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Department of Integrative Medical Sciences
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Title
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Role of SDF:CXCR4 in diet-induced cardiac injury
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Faseb Journal
Date
A point or period of time associated with an event in the lifecycle of the resource
2020
2020-04
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Dong F;Patel K;Kiedrowski M;Penn M
Description
An account of the resource
Introduction Diabetic cardiomyopathy (DCM) is a major cardiovascular complication in patients with diabetes and is defined as ventricular dysfunction (in diabetes) independent of coronary artery disease. In this study, we define a novel role for the SDF‐1: CXCR4 axis in diabetes‐associated myocardial dysfunction. Methods Wild‐type mice were randomly assigned to a high fat high sugar diet (HFHS) or control diet (LF) for 14 months. Serial echocardiography was used to assess cardiac function. The hSDF‐1 plasmid was injected into the LV wall of HFHS mice 7 months and 14 months after HFHS diet. The mRNA levels in the hearts were quantified by qPCR. Results HFHS‐fed mice (vs. control diet) showed significantly increased deceleration time (diastolic dysfunction) 7 months after HFHS diet and decreased Ejection Fraction (EF) (systolic dysfunction) 14 months after the HFHS diet. We observed a significant increase in cardiac myocytes surface area and a decrease in vascular density in the hearts of HFHS mice. HFHS‐fed mice showed decreased P‐selectin, E‐selectin expression and increased CXCR4, Resistin, and DDR2 expression in the heart. There is no significant improvement in diastolic function after the SDF‐1 injection at 7 month. However, direct myocardial injection of hSDF‐1 plasmid led to a significant improvement in EF compared to the control group. To determine the role of CM CXCR4 in hyperglycemia associated cardiac injury, we quantified cardiac function and survival rate in HFHS‐fed CM‐CXCR4 null mice. We observed a significantly increased mortality rate before 14 months on HFHS fed CM‐CXCR4 null mice compare to HFHS‐fed control mice. Conclusion HFHS diet induces diastolic dysfunction in the short term then systolic dysfunction after a long term. SDF‐1 treatment ameliorates systolic dysfunction but not diastolic dysfunction. Our data suggest that cardiac myocyte expression of CXCR4 has an important role in diabetes‐associated cardiac injury.
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<a href="http://doi.org/10.1096/fasebj.2020.34.s1.01787" target="_blank" rel="noreferrer noopener">10.1096/fasebj.2020.34.s1.01787</a>
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journalArticle
2020
Department of Integrative Medical Sciences
Dong F
Faseb Journal
journalArticle
Kiedrowski M
NEOMED College of Medicine
NEOMED College of Medicine Student
NEOMED Student Publications
Patel K
Penn M
September 2020 List
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
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<a href="http://doi.org/10.1096/fasebj.2020.34.s1.02623" target="_blank" rel="noreferrer noopener">http://doi.org/10.1096/fasebj.2020.34.s1.02623</a>
Issue
1
Volume
34
ISSN
0892-6638
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<a href="http://neomed.idm.oclc.org/login?url=http://doi.org/10.1096/fasebj.2020.34.s1.02623" target="_blank" rel="noreferrer noopener">NEOMED Full-text Holding (if available) - Proxy DOI: 10.1096/fasebj.2020.34.s1.02623</a>
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NEOMED College
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Department of Integrative Medical Sciences
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Title
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Molecular basis of takotsubo syndrome
Publisher
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Faseb Journal
Date
A point or period of time associated with an event in the lifecycle of the resource
2020
2020-04
Creator
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Chandler S;Joshi H;Hoff E;Patel K;Ohanyan V;Kiedrowski M;Chilian W;Dong F
Description
An account of the resource
Introduction Takotsubo syndrome (TTS), also known as the “Broken Heart Syndrome” or “Apical Ballooning Syndrome is defined by its characteristic anomaly: when the heart contracts during systole, the apex of the heart dilates as the base of the heart contracts. Severe TTS can lead to cardiogenic shock and death in 3–4% of patients. There is no standard medical therapy for TTS because the mechanism underlying the development of the syndrome is unknown. Our goal is to determine the molecular mechanisms of TTS as a first step towards better treatment plans and outcomes. Methods Our model of TTS is a Kv1.5 null mouse, with compromised coronary metabolic dilation, subjected to transaortic constriction (TAC). Two weeks after TAC, when Kv1.5 null mouse showed profound apical ballooning during ventricular contraction (echocardiography), myocardial blood flow (MBF) was measured by contrast echocardiography in the base and apex of the left ventricle of mice under control conditions and during acute administration of norepinephrine to increase cardiac work. Hearts were collected and gene expression (RNA deep sequencing) in both the apex and the base were performed and followed by bioinformatic analysis. Wild type (WT) and unstressed Kv1.5 null mice were used as controls. To increase the scientific rigor, we purposefully analyzed RNA expression from different animals with Real‐Time‐PCR, to confirm the sequencing data. Results A total of 3875 genes were identified by differentially expressed between TTS vs unstressed Kv1.5 null hearts. Gene Set Enrichment Analysis shows many families of genes downregulated (metabolism) and upregulated (inflammation, hypoxia, apoptosis) in the apical (ballooning) area of the heart in TTS (compared to the base of the heart). RT‐qPCR revealed significant upregulation of the genes for Postn, Lox, and C920009B18Rik (p<0.01) and down‐regulation of the genes Ucp3, Acaa2, Pfkb1, Mir133a‐2 (p<0.05). These significant changes in expression were also shown in the apex of TTS hearts as compared to the apex of control Kv1.5 null hearts (p<0.01). We also found that in the mice with TTS, MBF was lower in the apex than in the base. Conclusion Our results suggest that the apex of the heart in TTS is receiving insufficient perfusion compared to the base, which may be one of the root problems in TTS. There is significant upregulation of structural genes in the apex of TTS hearts versus their bases and the apexes of normal hearts, whereas there is a significant downregulation of genes playing roles in metabolism. This confirms the importance of these specific genes in the development of the anomaly shown in TTS.
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<a href="http://doi.org/10.1096/fasebj.2020.34.s1.02623" target="_blank" rel="noreferrer noopener">10.1096/fasebj.2020.34.s1.02623</a>
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journalArticle
2020
Chandler S
Chilian W
Department of Integrative Medical Sciences
Dong F
Faseb Journal
Hoff E
Joshi H
journalArticle
Kiedrowski M
NEOMED College of Medicine
NEOMED College of Medicine Student
NEOMED Student Publications
Ohanyan V
Patel K
September 2020 List
-
Text
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n/a
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
1-1
Issue
22
Volume
128
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Title
A name given to the resource
Early Up-regulation Of Myocardial Cxcr4 Expression Is Critical For Cardiac Improvement With Chemical Preconditioning In Acute Myocardial Infarction
Publisher
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Circulation
Date
A point or period of time associated with an event in the lifecycle of the resource
2013
2013-11
Subject
The topic of the resource
Cardiac regeneration; Cardiovascular System & Cardiology; heart failure; Myocardial infarction
Creator
An entity primarily responsible for making the resource
Kamath M; Kiedrowski M; Weber K; Forudi F; Penn M S; Dong F
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n/a
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Journal Article or Conference Abstract Publication
2013
Cardiac regeneration
Cardiovascular System & Cardiology
Circulation
Dong F
Forudi F
Heart failure
Journal Article or Conference Abstract Publication
Kamath M
Kiedrowski M
myocardial infarction
Penn M S
Weber K
-
Text
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<a href="http://doi.org/10.3109/14653249.2012.684380" target="_blank" rel="noreferrer noopener">http://doi.org/10.3109/14653249.2012.684380</a>
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
983-993
Issue
8
Volume
14
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Title
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Cardiac Pressure Overload Initiates A Systemic Stem Cell Response
Publisher
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Cytotherapy
Date
A point or period of time associated with an event in the lifecycle of the resource
2012
2012-09
Subject
The topic of the resource
& Experimental Medicine; acute myocardial-infarction; Biotechnology & Applied Microbiology; bone marrow; bone marrow; cardiac stem cells; cardiomyocytes; Cell Biology; endogenous stem cells; endothelial; endothelial progenitor cells; heart; Hematology; hypertrophy; identification; murine; peripheral-blood; progenitor cells; regeneration; Research; spleen; SSEA-1; transaortic constriction; transplantation
Creator
An entity primarily responsible for making the resource
Finan A; Kiedrowski M; Turturice B A; Sopko N A; Penn M S
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<a href="http://doi.org/10.3109/14653249.2012.684380" target="_blank" rel="noreferrer noopener">10.3109/14653249.2012.684380</a>
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The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
& Experimental Medicine
2012
acute myocardial-infarction
Biotechnology & Applied Microbiology
bone marrow
cardiac stem cells
cardiomyocytes
Cell Biology
Cytotherapy
endogenous stem cells
Endothelial
endothelial progenitor cells
Finan A
heart
Hematology
Hypertrophy
identification
Kiedrowski M
murine
Penn M S
peripheral-blood
progenitor cells
Regeneration
Research
Sopko N A
spleen
SSEA-1
transaortic constriction
Transplantation
Turturice B A
-
Text
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URL Address
<a href="http://doi.org/10.3727/096368913x670921" target="_blank" rel="noreferrer noopener">http://doi.org/10.3727/096368913x670921</a>
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
1395-1406
Issue
11
Volume
23
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Title
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Rat Mesenchymal Stem Cell Secretome Promotes Elastogenesis And Facilitates Recovery From Simulated Childbirth Injury
Publisher
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Cell Transplantation
Date
A point or period of time associated with an event in the lifecycle of the resource
2014
2014
Subject
The topic of the resource
Cell Biology; Elastin; Electromyography (EMG); expression; External urethral sphincter (EUS); female; female rats; mice; Paracrine factors; protection; Research & Experimental Medicine; Stress urinary incontinence (SUI); stress urinary-incontinence; stromal cells; tissue; transplantation; transplantation; Urethra; urethral sphincter; vaginal distension
Creator
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Dissaranan C; Cruz M A; Kiedrowski M; Balog B M; Gill B C; Penn M S; Goldman H B; Damaser M S
Identifier
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<a href="http://doi.org/10.3727/096368913x670921" target="_blank" rel="noreferrer noopener">10.3727/096368913x670921</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2014
Balog B M
Cell Biology
Cell Transplantation
Cruz M A
Damaser M S
Dissaranan C
elastin
Electromyography (EMG)
expression
External urethral sphincter (EUS)
Female
female rats
Gill B C
Goldman H B
Kiedrowski M
Mice
Paracrine factors
Penn M S
protection
Research & Experimental Medicine
Stress urinary incontinence (SUI)
stress urinary-incontinence
stromal cells
tissue
Transplantation
urethra
urethral sphincter
vaginal distension
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
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n/a
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
956-958
Issue
6
Volume
31
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Title
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Mesenchymal Stem Cells Facilitate Pudendal Nerve Recovery From Simulated Childbirth Injury
Publisher
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Neurourology and Urodynamics
Date
A point or period of time associated with an event in the lifecycle of the resource
2012
2012-08
Subject
The topic of the resource
Urology & Nephrology
Creator
An entity primarily responsible for making the resource
Damaser M; Lin D L; Hanzlicek B; Kiedrowski M; Balog B; Penn M; Goldman H
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n/a
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Journal Article or Conference Abstract Publication
2012
Balog B
Damaser M
Goldman H
Hanzlicek B
Kiedrowski M
Lin D L
Neurourology and Urodynamics
Penn M
Urology & Nephrology
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
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n/a
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
162-163
Volume
8
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Title
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Mesenchymal Stem Cells Or Their Secretome Promote Recovery And Alter Urethral Elastin In An Animal Model Of Childbirth Injuries
Publisher
An entity responsible for making the resource available
Journal of Tissue Engineering and Regenerative Medicine
Date
A point or period of time associated with an event in the lifecycle of the resource
2014
2014-06
Subject
The topic of the resource
Biotechnology & Applied Microbiology; Cell Biology; Engineering
Creator
An entity primarily responsible for making the resource
Damaser M; Deng K; Lin D L; Hanzlicek B; Balog B; Penn M; Kiedrowski M; Zhu H
Identifier
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n/a
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2014
Balog B
Biotechnology & Applied Microbiology
Cell Biology
Damaser M
Deng K
Engineering
Hanzlicek B
Journal of Tissue Engineering and Regenerative Medicine
Kiedrowski M
Lin D L
Penn M
Zhu H
-
Text
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URL Address
<a href="http://doi.org/10.1016/j.jsxm.2019.01.194" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/j.jsxm.2019.01.194</a>
Pages
S92–S92
Issue
4
Volume
16
ISSN
1743-6095
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Title
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IMPACT OF RHOGDI GENE TRANSFECTION OF BLADDER SMOOTH MUSCLE CONTRACTILITY IN A VALIDATED EX-VIVO MURINE MODEL
Publisher
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Journal of Sexual Medicine
Date
A point or period of time associated with an event in the lifecycle of the resource
2019
2019-04
Subject
The topic of the resource
Urology & Nephrology
Creator
An entity primarily responsible for making the resource
Joice G; Bell J M; La Favor J; Yoshida T; Torga G; Harris K; Liu X; Kiedrowski M; Penn M; Bivalacqua T
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<a href="http://doi.org/10.1016/j.jsxm.2019.01.194" target="_blank" rel="noreferrer noopener">10.1016/j.jsxm.2019.01.194</a>
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An account of the resource
19th Annual Fall Scientific Meeting of SMSNA
Introduction Plasmid-based gene therapy is an intriguing option for treating malignant and bladder pathologies. The RhoA pathway is involved in bladder smooth muscle regulation, cancer invasion and metastasis. Rho GDP-dissociation inhibitor (RhoGDI) is an inhibitor of the RhoA pathway. We validated ex-vivo bladder gene transfer to facilitate assessment of gene targets for treating bladder pathology. Methods Basic Local Alignment Search Tool was used to identify human RhoGDI coding sequences and to compare between rats and humans, which were cloned into a eCMV-based expression vector. NBTII rat bladder cancer cell lines were transfected using FuGENE (Promega, USA) and human protein expression and interaction with endogenous RhoA were tested using flow cytometry, immunofluorescence, and RNA expression analysis. Bladders were harvested from female Lewis Rats (∼250g) and sectioned and cultured for 72-hours following transfection with RhoGDI and FuGENE. Transfected bladder tissues were analyzed as described above. Non-transfected cultured bladder segments were analyzed using myography for viability and intact smooth muscle physiology in response to 120 mM KCl and 30uM carbachol. Results Human and rodent RhoGDI protein homology is 96%. Human RhoGDI was successfully detected exclusively in transfected NBTII cells with a top efficiency of 26%. qPCR analysis demonstrated rodent RhoA and RhoGDI levels were not impacted but ROCK1 and ROCK2 mRNA were significantly decreased by 23.6% (p=0.034) and 40.0% (p=0.015), respectively following human RhoGDI transfection. Human RhoGDI was detected in transfected ex-vivo cultured bladder segments in both FuGENE and microinjection experiments. Similar to NBTII cells, qPCR analysis demonstrated rodent RhoA and RhoGDI levels were not impacted but ROCK1 and ROCK2 mRNA were significantly decreased by 15.0% (p=0.035) and 22.4% (p=0.010) after FuGENE transfection and 20.5% (p=0.024) and 21.4% (p=0.015) after microinjections. Ex-vivo cultured bladder strips successfully contracted to KCl (mean 0.88+/-0.48 mN/mg tissue) and carbachol (1.82+/-0.97 mN/mg tissue) stimulation. After microinjection, RhoGDI caused a significant reduction in KCl mediated constriction although this was not observed in FuGENE experiments. Conclusion Ex-vivo bladder culture, transfection, and physiological assessment are feasible and may provide a high-throughput method to test novel gene transfer technologies before in-vivo testing. RhoGDI plasmid microinjection transfection appears to decrease contractility of ex-vivo bladder smooth muscle.
2019
Bell J M
Bivalacqua T
Department of Integrative Medical Sciences
Harris K
Joice G
Journal of Sexual Medicine
June 2019 Update
Kiedrowski M
La Favor J
Liu X
NEOMED College of Medicine
Penn M
Torga G
Urology & Nephrology
Yoshida T
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="https://doi.org/10.1016/j.jsxm.2019.01.194">https://doi.org/10.1016/j.jsxm.2019.01.194</a>
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Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
S92-S92
Issue
4
Volume
16
Dublin Core
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Title
A name given to the resource
185 Impact of RhoGDI Gene Transfection of Bladder Smooth Muscle Contractility in a Validated Ex-vivo Murine Model
Publisher
An entity responsible for making the resource available
The Journal of Sexual Medicine
Date
A point or period of time associated with an event in the lifecycle of the resource
2019
2019-04
Creator
An entity primarily responsible for making the resource
Joice G; Bell J M; La Favor J; Yoshida T; Torga G; Harris K; Liu X; Kiedrowski M; Penn M; Bivalacqua T
Identifier
An unambiguous reference to the resource within a given context
<a href="https://doi.org/10.1016/j.jsxm.2019.01.194">https://doi.org/10.1016/j.jsxm.2019.01.194</a>
2019
Bell J M
Bivalacqua T
Department of Internal Medicine
Harris K
Joice G
June 2019 Update
Kiedrowski M
La Favor J
Liu X
NEOMED College of Medicine
Penn M
The Journal of Sexual Medicine
Torga G
Yoshida T