1
40
2
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1007/s100169910006" target="_blank" rel="noreferrer noopener">http://doi.org/10.1007/s100169910006</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
31-36
Issue
1
Volume
14
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Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Systemic consequences of oxidative stress following aortic surgery correlate with the degree of antioxidant defenses
Publisher
An entity responsible for making the resource available
Annals of Vascular Surgery
Date
A point or period of time associated with an event in the lifecycle of the resource
2000
2000-01
Subject
The topic of the resource
Surgery; oxygen; pulmonary-edema; Cardiovascular System & Cardiology; capacity; ischemia-reperfusion injury; radicals; lung injury; depletion; mediators; xanthine-oxidase
Creator
An entity primarily responsible for making the resource
Cornu-Labat G; Serra M; Smith A; McGregor W E; Kasirajan K; Hirko M K; Turner J J; Rubin J R
Description
An account of the resource
The purpose of this study was to correlate the preoperative level of antioxidant defenses, measured by the plasma total antioxidant capacity (TAC), to the degree of postoperative systemic inflammatory response, measured by the severity of pulmonary injury following elective aortic surgery. Twenty-four patients had TAC measured preoperatively and 24 hr postoperatively. Chest radiography and arterial blood gases were obtained preoperatively and serially during the first 24 hr after surgery. Using objective radiologic criteria and blood gas analysis, the degree of pulmonary edema and pulmonary dysfunction were quantified. All patients showed evidence of pulmonary dysfunction in the first 24 hr following surgery. Fifteen of the 24 patients showed radiographic evidence of noncardiogenic pulmonary edema in the immediate postoperative period. In this group, the TAC was lower than in those without pulmonary edema immediately following surgery (p = 0.03). Preoperative TAC was associated with the degree of pulmonary edema in the postoperative period (r = -0.372, p = 0.067). These results suggest that preoperative antioxidant supplementation may favorably impact the severity of systemic inflammatory response following ischemia and reperfusion injury. DOI: 10.1007/s100169910006.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1007/s100169910006" target="_blank" rel="noreferrer noopener">10.1007/s100169910006</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2000
Annals of Vascular Surgery
capacity
Cardiovascular System & Cardiology
Cornu-Labat G
depletion
Hirko M K
ischemia-reperfusion injury
Journal Article or Conference Abstract Publication
Kasirajan K
Lung Injury
McGregor W E
mediators
Oxygen
pulmonary-edema
radicals
Rubin J R
Serra M
Smith A
Surgery
Turner J J
xanthine-oxidase
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1152/japplphysiol.01201.2001" target="_blank" rel="noreferrer noopener">http://doi.org/10.1152/japplphysiol.01201.2001</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
2207-2213
Issue
6
Volume
93
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
<p>Users with a NEOMED Library login can search for full-text journal articles at the following url: <a href="https://libraryguides.neomed.edu/home">https://libraryguides.neomed.edu/home</a></p>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Lung edema clearance: 20 years of progress - Invited review: Alveolar edema fluid clearance in the injured lung
Publisher
An entity responsible for making the resource available
Journal of Applied Physiology
Date
A point or period of time associated with an event in the lifecycle of the resource
2002
2002-12
Subject
The topic of the resource
therapy; liquid clearance; Physiology; pulmonary-edema; Sport Sciences; sodium transport; agonist; epithelial; active sodium-transport; alveolar fluid clearance; barrier function; agonists; respiratory-distress syndrome; na-k-atpase; adenovirus-mediated transfer; beta-adrenergic; beta(1) subunit gene; lung injury; rat lungs
Creator
An entity primarily responsible for making the resource
Berthiaume Y; Folkesson H G; Matthay M A
Description
An account of the resource
Resolution of pulmonary edema involved active transepithelial sodium transport. Although several of the cellular and molecular mechanisms involved are relatively well understood, it is only recently that the regulation of these mechanisms in injured lung are being evaluated. Interestingly, in mild-to-moderate lung injury, alveolar edema fluid clearance is often preserved. This preserved or enhanced alveolar fluid clearance is mediated by catecholamine-dependent or -independent mechanisms. This stimulation of alveolar liquid clearance is related to activation or increased expression of sodium transport molecules such as the epithelial sodium channel or the Na+-K+-ATPase pump and may also involve the cystic fibrosis transmembrane conductance regulator. When severe lung injury occurs, the decrease in alveolar liquid clearance may be related to changes in alveolar permeability or to changes in activity or expression of sodium or chloride transport molecules. Multiple pharmacological tools such as beta-adrenergic agonists, vasoactive drugs, or gene therapy may prove effective in stimulating the resolution of alveolar edema in the injured lung.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1152/japplphysiol.01201.2001" target="_blank" rel="noreferrer noopener">10.1152/japplphysiol.01201.2001</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2002
active sodium-transport
adenovirus-mediated transfer
agonist
agonists
alveolar fluid clearance
barrier function
Berthiaume Y
beta-adrenergic
beta(1) subunit gene
epithelial
Folkesson H G
Journal Article or Conference Abstract Publication
Journal of Applied Physiology
liquid clearance
Lung Injury
Matthay M A
na-k-atpase
Physiology
pulmonary-edema
rat lungs
respiratory-distress syndrome
sodium transport
Sport Sciences
therapy