1
40
1
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.3389/fped.2019.00263" target="_blank" rel="noreferrer noopener">http://doi.org/10.3389/fped.2019.00263</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
263-263
Volume
7
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
<p>Users with a NEOMED Library login can search for full-text journal articles at the following url: <a href="https://libraryguides.neomed.edu/home">https://libraryguides.neomed.edu/home</a></p>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Overview of Monogenic or Mendelian Forms of Hypertension
Publisher
An entity responsible for making the resource available
Frontiers in Pediatrics
Date
A point or period of time associated with an event in the lifecycle of the resource
2019
1905-07
Subject
The topic of the resource
aldosteronism; apparent mineralocorticoid excess; congenital adrenal hyperplasia; disease; familial; familial hyperaldosteronism; features; genes; Gordon syndrome; hyperaldosteronism; Liddle syndrome; Liddle syndrome; mineralocorticoid excess; monogenic hypertension; mutations; Pediatrics; prevalence; stroke; update
Creator
An entity primarily responsible for making the resource
Raina Rupesh; Krishnappa Vinod; Das Abhijit; Amin Harshesh; Radhakrishnan Yeshwanter; Nair Nikhil R; Kusumi Kirsten
Description
An account of the resource
Monogenic or Mendelian forms of hypertension are described as a group of conditions characterized by insults to the normal regulation of blood pressure by the kidney and adrenal gland. These alterations stem from single mutations that lead to maladaptive overabsorption of electrolytes with fluid shift into the vasculature, and consequent hypertension. Knowledge of these various conditions is essential in diagnosing pediatric or early-onset adult hypertension as they directly affect treatment strategies. Precise diagnosis with specific treatment regimens aimed at the underlying physiologic derangement can restore normotension and prevent the severe sequelae of chronic hypertension.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.3389/fped.2019.00263" target="_blank" rel="noreferrer noopener">10.3389/fped.2019.00263</a>
2019
aldosteronism
Amin Harshesh
apparent mineralocorticoid excess
congenital adrenal hyperplasia
Das Abhijit
Department of Internal Medicine
Disease
familial
familial hyperaldosteronism
features
Frontiers in pediatrics
Genes
Gordon syndrome
hyperaldosteronism
Krishnappa Vinod
Kusumi Kirsten
Liddle syndrome
mineralocorticoid excess
monogenic hypertension
mutations
Nair Nikhil R
NEOMED College of Graduate Studies Student
NEOMED College of Medicine
NEOMED College of Medicine Student
NEOMED Student Publications
Pediatrics
Prevalence
Radhakrishnan Yeshwanter
Raina Rupesh
September 2019 Update
stroke
update