1
40
5
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1152/japplphysiol.01014.2009" target="_blank" rel="noreferrer noopener">http://doi.org/10.1152/japplphysiol.01014.2009</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
1766-1774
Issue
6
Volume
108
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
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Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Short-term Exercise Training Prevents Micro-and Macrovascular Disease Following Coronary Stenting
Publisher
An entity responsible for making the resource available
Journal of Applied Physiology
Date
A point or period of time associated with an event in the lifecycle of the resource
2010
2010-06
Subject
The topic of the resource
aortic smooth-muscle; artery-disease; cholesterol-lowering therapy; diabetic dyslipidemic swine; dysfunction; endothelial; exercise; flow-velocity reserve; hyperlipidemia; low-fat diet; microvessel; myocardial-perfusion; ossabaw miniature swine; physical-exercise; Physiology; regular; restenosis; Sport Sciences; Yucatan swine
Creator
An entity primarily responsible for making the resource
Long X; Bratz I N; Alloosh M; Edwards J M; Sturek M
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1152/japplphysiol.01014.2009" target="_blank" rel="noreferrer noopener">10.1152/japplphysiol.01014.2009</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2010
Alloosh M
aortic smooth-muscle
artery-disease
Bratz I N
cholesterol-lowering therapy
diabetic dyslipidemic swine
dysfunction
Edwards J M
Endothelial
Exercise
flow-velocity reserve
Hyperlipidemia
Journal of Applied Physiology
Long X
low-fat diet
microvessel
myocardial-perfusion
Ossabaw miniature swine
physical-exercise
Physiology
regular
Restenosis
Sport Sciences
Sturek M
Yucatan swine
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1007/s00395-013-0387-4" target="_blank" rel="noreferrer noopener">http://doi.org/10.1007/s00395-013-0387-4</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
12-12
Issue
6
Volume
108
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
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Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Role Of Genetic Polymorphisms Of Ion Channels In The Pathophysiology Of Coronary Microvascular Dysfunction And Ischemic Heart Disease
Publisher
An entity responsible for making the resource available
Basic Research in Cardiology
Date
A point or period of time associated with an event in the lifecycle of the resource
2013
2013-11
Subject
The topic of the resource
artery-disease; atherosclerosis; Atrial fibrillation; Cardiovascular System & Cardiology; Coronary microcirculation; dysfunction; endothelial; Endothelium; gene; Genetic polymorphisms; Ion channels; Ischemic heart disease; k-atp channels; kir6.2; late sodium current; nitric-oxide; sensitive potassium channels; smooth-muscle; type-2 diabetes-mellitus; vascular
Creator
An entity primarily responsible for making the resource
Fedele F; Mancone M; Chilian W M; Severino P; Canali E; Logan S; De Marchis M L; Volterrani M; Palmirotta R; Guadagni F
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1007/s00395-013-0387-4" target="_blank" rel="noreferrer noopener">10.1007/s00395-013-0387-4</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2013
artery-disease
Atherosclerosis
Atrial fibrillation
Basic research in cardiology
Canali E
Cardiovascular System & Cardiology
Chilian W M
Coronary microcirculation
De Marchis M L
dysfunction
Endothelial
Endothelium
Fedele F
gene
Genetic polymorphisms
Guadagni F
Ion Channels
Ischemic heart disease
k-atp channels
kir6.2
late sodium current
Logan S
Mancone M
nitric-oxide
Palmirotta R
sensitive potassium channels
Severino P
smooth-muscle
type-2 diabetes-mellitus
Vascular
Volterrani M
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1038/srep03274" target="_blank" rel="noreferrer noopener">http://doi.org/10.1038/srep03274</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
6-6
Volume
3
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
<p>Users with a NEOMED Library login can search for full-text journal articles at the following url: <a href="https://libraryguides.neomed.edu/home">https://libraryguides.neomed.edu/home</a></p>
Dublin Core
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Title
A name given to the resource
Cysteinyl Leukotrienes Regulate Endothelial Cell Inflammatory And Proliferative Signals Through Cyslt(2) And Cyslt(1) Receptors
Publisher
An entity responsible for making the resource available
Scientific Reports
Date
A point or period of time associated with an event in the lifecycle of the resource
2013
2013-11
Subject
The topic of the resource
atherosclerosis; barrier; dysfunction; expression; gamma; intestinal epithelial-cells; ischemia-reperfusion injury; migration; pathway; Science & Technology - Other Topics; thrombin
Creator
An entity primarily responsible for making the resource
Duah E; Adapala R K; Al-Azzam N; Kondeti V; Gombedza F; Thodeti C K; Paruchuri S
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1038/srep03274" target="_blank" rel="noreferrer noopener">10.1038/srep03274</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2013
Adapala R K
Al-Azzam N
Atherosclerosis
barrier
Duah E
dysfunction
expression
gamma
Gombedza F
intestinal epithelial-cells
ischemia-reperfusion injury
Kondeti V
migration
Paruchuri S
pathway
Science & Technology - Other Topics
Scientific reports
Thodeti C K
Thrombin
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1152/ajpheart.00694.2010" target="_blank" rel="noreferrer noopener">http://doi.org/10.1152/ajpheart.00694.2010</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
H1069-H1077
Issue
3
Volume
300
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
<p>Users with a NEOMED Library login can search for full-text journal articles at the following url: <a href="https://libraryguides.neomed.edu/home">https://libraryguides.neomed.edu/home</a></p>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Endothelial NOS activity and myocardial oxygen metabolism define the salvageable ischemic time window for ischemic postconditioning
Publisher
An entity responsible for making the resource available
American Journal of Physiology-Heart and Circulatory Physiology
Date
A point or period of time associated with an event in the lifecycle of the resource
2011
2011-03
Subject
The topic of the resource
cardioprotection; mice; inhibition; Physiology; Cardiovascular System & Cardiology; activation; dysfunction; reperfusion injury; tissue oxygenation; consumption; blood flow; electron paramagnetic resonance; infarct size; ischemia and reperfusion; mitochondrial enzyme activity; mitochondrial permeability transition; rabbit hearts; regional
Creator
An entity primarily responsible for making the resource
Cai M; Li Y J; Xu Y; Swartz H M; Chen C L; Chen Y R; He G L
Description
An account of the resource
Cai M, Li Y, Xu Y, Swartz HM, Chen C, Chen Y, He G. Endothelial NOS activity and myocardial oxygen metabolism define the salvageable ischemic time window for ischemic postconditioning. Am J Physiol Heart Circ Physiol 300: H1069-H1077, 2011. First published January 7, 2011; doi: 10.1152/ajpheart.00694.2010.-Ischemic postconditioning (IPOC) could be ineffective or even detrimental if the index ischemic duration is either too short or too long. The present study is to demonstrate that oxygen supply and metabolism defines a salvageable ischemic time window of IPOC in mice. C57BL/6 mice underwent coronary artery occlusion followed by reperfusion (I/R), with or without IPOC by three cycles of 10 s/10 s R/I. In vivo myocardial tissue oxygenation was monitored with electron paramagnetic resonance oximetry. Regional blood flow (RBF) was measured with a laser Doppler monitor. At the end of 60 min reperfusion, tissue from the risk area was collected, and mitochondrial enzyme activities were assayed. Tissue oximetry demonstrated that I/R induced a reperfusion hyperoxygenation state in the 30- and 45-min but not 15- and 60-min ischemia groups. IPOC attenuated the hyperoxygenation with 45 but not 30 min ischemia. RBF, eNOS phosphorylation, and mitochondrial enzyme activities were suppressed after I/R with different ischemic time, and IPOC afforded protection with 30 and 45 but not 60 min ischemia. Infarct size measurement indicated that IPOC reduced infarction with 30 and 45 min but not 60 min ischemia. Clearly, IPOC protected mouse heart with a defined ischemic time window between 30 and 45 min. This salvageable time window was accompanied by the improvement of RBF due to increased phosphorylated eNOS and the preservation of mitochondrial oxygen consumption due to conserved mitochondrial enzyme activities. Interestingly, this salvageable ischemic time window was mirrored by tissue hyperoxygenation status in the postischemic heart.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1152/ajpheart.00694.2010" target="_blank" rel="noreferrer noopener">10.1152/ajpheart.00694.2010</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2011
activation
American Journal of Physiology-Heart and Circulatory Physiology
blood flow
Cai M
cardioprotection
Cardiovascular System & Cardiology
Chen C L
Chen Y R
consumption
dysfunction
electron paramagnetic resonance
He G L
infarct size
inhibition
ischemia and reperfusion
Journal Article or Conference Abstract Publication
Li Y J
Mice
mitochondrial enzyme activity
mitochondrial permeability transition
Physiology
rabbit hearts
regional
Reperfusion Injury
Swartz H M
tissue oxygenation
Xu Y
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1001/archfami.8.5.440" target="_blank" rel="noreferrer noopener">http://doi.org/10.1001/archfami.8.5.440</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
440-444
Issue
5
Volume
8
Search for Full-text
Locate full-text within NEOMED Library's e-journal collections
<p>Users with a NEOMED Library login can search for full-text journal articles at the following url: <a href="https://libraryguides.neomed.edu/home">https://libraryguides.neomed.edu/home</a></p>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Physicians' attention to parents' concerns about the psychosocial functioning of their children
Publisher
An entity responsible for making the resource available
Archives of Family Medicine
Date
A point or period of time associated with an event in the lifecycle of the resource
1999
1999-09
Subject
The topic of the resource
depression; dysfunction; epidemiology; General & Internal Medicine; Health; management; pediatric primary care; prevalence; psychiatric-disorders; psychopathology; service utilization
Creator
An entity primarily responsible for making the resource
Wildman B G; Kizilbash A H; Smucker W D
Description
An account of the resource
Background: Epidemiological data indicate that approximately 20% of children have psychosocial problems, yet less than 2% of children are seen by mental health specialists each year. Primary care physicians tend to identify children with psychosocial problems when parents discuss concerns with them. Objective: To examine factors related to physicians' attention to parental disclosures. Design: Parents reported the psychosocial functioning of themselves and their children. Physicians reported the psychosocial functioning of 75; children and whether the parent disclosed psychosocial concerns to them. Setting: Ambulatory care clinic of a community-based, university-affiliated, residency training program. Participants: Seventy-five parents of children aged 2 to 16 years who presented for routine primary care, and 26 physicians. Main Outcome Measures: Beck Depression Inventory (parental distress), Eyberg Child Behavior Inventory (child behavior problems) , physician and parent report. Results: Physicians identified 50.0% of children with clinically significant behavior problems. Logistic regression indicated that parental disclosure was the only significant predictor of physician identification (P<.002). When children had clinically significant behavior problems, physicians were more likely to report disclosures by parents (45.0% vs 5.7% for parents of children with and without behavior problems, respectively). Physicians were more likely to report parental disclosure when parents reported personal psychosocial distress (38.9% for distressed vs 5.7% nondistressed parents). Conclusions: Parental disclosure of concerns was a better predictor of physician identification of child psychosocial problems than was the presence of child behavior problems. Physicians responded more frequently to the disclosures of potential problems by parents of children with clinically significant psychosocial problems. They also attended more frequently to disclosures about behavior problems when the parent was also experiencing psychosocial distress.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1001/archfami.8.5.440" target="_blank" rel="noreferrer noopener">10.1001/archfami.8.5.440</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article
1999
Archives of family medicine
Department of Family & Community Medicine
Depression
dysfunction
Epidemiology
General & Internal Medicine
Health
Journal Article
Kizilbash A H
Management
NEOMED College of Medicine
pediatric primary care
Prevalence
psychiatric-disorders
psychopathology
service utilization
Smucker W D
Wildman B G