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Text
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URL Address
<a href="http://doi.org/10.1358/dof.2010.35.8.1520865" target="_blank" rel="noreferrer noopener">http://doi.org/10.1358/dof.2010.35.8.1520865</a>
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Pages
635-641
Issue
8
Volume
35
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Title
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FARNESOID X RECEPTOR: ACTING THROUGH BILE ACIDS TO TREAT METABOLIC DISORDERS
Publisher
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Drugs of the Future
Date
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2010
2010-08
Subject
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fatty liver-disease; foam-cell formation; growth-factor receptor; heterodimer partner; insulin-resistance; nonalcoholic steatohepatitis; orphan nuclear receptor; Pharmacology & Pharmacy; primary rat hepatocytes; protein-kinase-c; regulatory cascade; small
Creator
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Zhang Y
Description
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The famesoid X receptor (FXR) is a member of the nuclear receptor superfamily and plays an important role in maintaining bile acid, lipid and glucose homeostasis. Bile acids are endogenous ligands for FXR. However, bile acids may also activate pathways independent of FXR. The development of specific FXR agonists has provided important insights into the role of FXR in metabolism. Recent data have demonstrated that FXR is a therapeutic target for the treatment of certain metabolic disorders. This review will focus on recent advances in the role of FXR in metabolic disease.
Identifier
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<a href="http://doi.org/10.1358/dof.2010.35.8.1520865" target="_blank" rel="noreferrer noopener">10.1358/dof.2010.35.8.1520865</a>
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Journal Article
2010
Drugs of the future
fatty liver-disease
foam-cell formation
growth-factor receptor
heterodimer partner
insulin-resistance
Journal Article
Nonalcoholic steatohepatitis
orphan nuclear receptor
Pharmacology & Pharmacy
primary rat hepatocytes
protein-kinase-c
regulatory cascade
Small
Zhang Y