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40
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Text
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URL Address
<a href="http://doi.org/10.1016/j.ekir.2020.03.025" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/j.ekir.2020.03.025</a>
Pages
980-990
Issue
7
Volume
5
ISSN
2468-0249
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<a href="http://neomed.idm.oclc.org/login?url=http://doi.org/10.1016/j.ekir.2020.03.025" target="_blank" rel="noreferrer noopener">NEOMED Full-text Holding (if available) - Proxy DOI: 10.1016/j.ekir.2020.03.025</a>
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Update Year & Number
August 2020 List
NEOMED College
NEOMED College of Medicine
NEOMED Department
Department of Internal Medicine
Department of Anatomy & Neurobiology
NEOMED Student Publications
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Title
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An overview of rickets in children
Publisher
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Kidney International Reports
Date
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2020
2020-07
Subject
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prevention; vitamin D; mutations; animal-model; phosphate; chronic kidney disease; phosphorus; d-receptor; d-resistant rickets; hereditary hypophosphatemic rickets; hypocalcemia; hypophosphatemia; targeted ablation; vitamin-d-deficiency; x-linked hypophosphatemia
Creator
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Chanchlani R; Nemer P; Sinha R; Nemer L; Krishnappa V; Sochett E; Safadi F; Raina R
Description
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Rickets is a common bone disease worldwide that is associated with disturbances in calcium and phos- phate homeostasis and can lead to short stature and joint deformities. Rickets can be diagnosed based on history and physical examination, radiological features, and biochemical tests. It can be classified into 2 major groups based on phosphate or calcium levels: phosphopenic and calcipenic. Knowledge of cate- gorization of the type of rickets is essential for prompt diagnosis and proper management. Nutritional rickets is a preventable disease through adequate intake of vitamin D through both dietary and sunlight exposure. There are other subtypes of rickets, such as vitamin D-dependent type 1 rickets and vitamin D- dependent type 2 rickets (due to defects in vitamin D metabolism), renal rickets (due to poor kidney function), and hypophosphatemic rickets (vitamin D-resistant rickets secondary to renal phosphate wasting wherein fibroblast growth factor -23 (FGF-23) often plays a major role), which requires closer monitoring and supplementation with activated vitamin D with or without phosphate supplements. An important development has been the introduction of burosumab, a human monoclonal antibody to FGF- 23, which is approved for the treatment of X -linked hypophosphatemia among children 1 year and older.
Identifier
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<a href="http://doi.org/10.1016/j.ekir.2020.03.025" target="_blank" rel="noreferrer noopener">10.1016/j.ekir.2020.03.025</a>
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journalArticle
2020
animal-model
August 2020 List
Chanchlani R
Chronic kidney disease
d-receptor
d-resistant rickets
Department of Anatomy & Neurobiology
Department of Internal Medicine
hereditary hypophosphatemic rickets
hypocalcemia
hypophosphatemia
journalArticle
Kidney International Reports
Krishnappa V
mutations
Nemer L
Nemer P
NEOMED College of Medicine
NEOMED College of Medicine Student
phosphate
phosphorus
Prevention
Raina R
Safadi F
Sinha R
Sochett E
targeted ablation
Vitamin D
vitamin-d-deficiency
x-linked hypophosphatemia