1
40
12
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<table width="91" style="border-collapse:collapse;width:68pt;"><colgroup><col width="91" style="width:68pt;" /></colgroup><tbody><tr style="height:15pt;"><td width="91" height="20" class="xl18" style="width:68pt;height:15pt;"><a href="http://doi.org/10.7759/cureus.14084">http://doi.org/10.7759/cureus.14084</a></td>
</tr></tbody></table>
NEOMED College
NEOMED College of Medicine
NEOMED Department
Department fo Internal Medicine
Update Year & Number
Jan to Aug list 2021
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Long-Term Sequela of Intrathecal Gadolinium Extravasation: Symptoms Mimicking Post-concussive Syndrome
Creator
An entity primarily responsible for making the resource
Patel A; Zolyan A; Itrat A
Publisher
An entity responsible for making the resource available
Cureus
Date
A point or period of time associated with an event in the lifecycle of the resource
2021
2021-03-24
Description
An account of the resource
Gadolinium contrast administration, usually with magnetic resonance imaging, is an important diagnostic modality in the investigation of neurological pathologies. There is little evidence in the literature suggesting repeated exposure to intrathecal gadolinium results in symptoms mimicking post-concussive syndrome (PCS). We studied one patient who received intrathecal gadolinium to investigate a pain pump malfunction and presented with encephalopathic symptoms of confusion and aphasia with imaging consistent with intracranial gadolinium extravasation. The patient was followed up regularly with repeat imaging, reassessment of persistent symptoms, and specialist evaluations; however, symptoms remained refractory and resembled PCS. Our findings indicate a need to further investigate potential associations between intrathecal gadolinium exposure and a clinical presentation consistent with PCS, irrespective of histopathological changes.
Subject
The topic of the resource
Repeated exposure to intravenous gadolinium is thought to have a cumulative effect over time with brain deposits that have been histopathologically confirmed in patients with normal renal function. Acute extravasation of gadolinium contents mimicking acute subarachnoid hemorrhage has also been described elsewhere, especially after intrathecal administration, including the patient in our case report [1,2]. There is, however, no clear consensus yet if these acute “high-dose” or gradually accumulating deposits result in long-term neurological sequelae. Recent studies have demonstrated gadolinium toxicity in preclinical in-vitro studies by pathophysiological mechanisms involving calcium homeostasis [3]. Once within the extracellular space, gadolinium is proposed to have affinity for certain other similar metals with resulting chelation and deposition in the central nervous system [4]. Animal studies have demonstrated increased markers of cellular injury after exposure to higher concentrations of gadolinium [5]. However, causal relationship is difficult to establish between gadolinium administration and neurological sequela [3]. We had the unique opportunity to study one such patient with acute toxic encephalopathy over a period of two years and report subsequent clinical findings.
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<table width="91" style="border-collapse:collapse;width:68pt;"><colgroup><col width="91" style="width:68pt;" /></colgroup><tbody><tr style="height:15pt;"><td width="91" height="20" class="xl18" style="width:68pt;height:15pt;"><a href="http://doi.org/10.7759/cureus.14084">http://doi.org/10.7759/cureus.14084</a></td>
</tr></tbody></table>
Format
The file format, physical medium, or dimensions of the resource
Journal Article
2021
gadolinium
Neurology
neuroradiology
post-concussive syndrome
Radiology
toxic encephalopathy
toxicity
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.7759/cureus.14084" target="_blank" rel="noreferrer noopener">http://doi.org/10.7759/cureus.14084</a>
Issue
3
Volume
13
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<a href="http://neomed.idm.oclc.org/login?url=http://doi.org/10.7759/cureus.14084" target="_blank" rel="noreferrer noopener">NEOMED Full-text Holding (if available) - Proxy DOI: 10.7759/cureus.14084</a>
<p>Users with a NEOMED Library login can search for full-text journal articles at the following url: <a href="https://libraryguides.neomed.edu/home">https://libraryguides.neomed.edu/home</a></p>
Update Year & Number
May 2021 List
NEOMED College
NEOMED College of Medicine
NEOMED Department
NEOMED Student Publications
Department fo Internal Medicine
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Long-Term Sequela of Intrathecal Gadolinium Extravasation: Symptoms Mimicking Post-concussive Syndrome
Publisher
An entity responsible for making the resource available
Cureus
Date
A point or period of time associated with an event in the lifecycle of the resource
2021
2021-03-24
Subject
The topic of the resource
toxicity; neurology; radiology; gadolinium; neuroradiology; post-concussive syndrome; toxic encephalopathy
Creator
An entity primarily responsible for making the resource
Patel A; Zolyan A; Itrat A
Description
An account of the resource
Gadolinium contrast administration, usually with magnetic resonance imaging, is an important diagnostic modality in the investigation of neurological pathologies. There is little evidence in the literature suggesting repeated exposure to intrathecal gadolinium results in symptoms mimicking post-concussive syndrome (PCS). We studied one patient who received intrathecal gadolinium to investigate a pain pump malfunction and presented with encephalopathic symptoms of confusion and aphasia with imaging consistent with intracranial gadolinium extravasation. The patient was followed up regularly with repeat imaging, reassessment of persistent symptoms, and specialist evaluations; however, symptoms remained refractory and resembled PCS. Our findings indicate a need to further investigate potential associations between intrathecal gadolinium exposure and a clinical presentation consistent with PCS, irrespective of histopathological changes.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.7759/cureus.14084" target="_blank" rel="noreferrer noopener">10.7759/cureus.14084</a>
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Information about rights held in and over the resource
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Format
The file format, physical medium, or dimensions of the resource
journalArticle
2021
Cureus
Department of Internal Medicine
gadolinium
Itrat A
journalArticle
May 2021 List
NEOMED College of Medicine
NEOMED Student Publications
Neurology
neuroradiology
Patel A
post-concussive syndrome
Radiology
toxic encephalopathy
toxicity
Zolyan A
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1016/j.brainres.2012.10.029" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/j.brainres.2012.10.029</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
133-139
Volume
1489
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Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Ngp1-01, A Multi-targeted Polycyclic Cage Amine, Attenuates Brain Endothelial Cell Death In Iron Overload Conditions
Publisher
An entity responsible for making the resource available
Brain Research
Date
A point or period of time associated with an event in the lifecycle of the resource
2012
2012-12
Subject
The topic of the resource
activator; barrier; calcium channels; channels provide; intracerebral hemorrhage; Iron-overload; neurodegeneration; neurodegenerative disorders; neuroprotection; Neurosciences & Neurology; Nimodipine; parkinsons-disease; permeability; rat-brain; toxicity; transport; Vascular endothelial cells; Voltage-gated calcium channel
Creator
An entity primarily responsible for making the resource
Lockman J A; Geldenhuys W J; Jones-Higgins M R; Patrick J D; Allen D D; Van der Schyf C J
Description
An account of the resource
Development and progression of neurodegenerative disorders have, amongst other potential causes, been attributed to a disruption of iron regulatory mechanisms and iron accumulation. Excess extracellular iron may enter cells via nontraditional routes such as voltage-gated calcium channels and N-methyl-D-aspartate (NMDA) receptors leading to intracellular oxidative damage and ultimately mitochondrial failure. Nimodipine, an L-type calcium channel blocker has been shown to reduce iron-induced toxicity in neuronal and brain endothelial cells. Our current study investigates NGP1-01, a multimodal drug acting as an antagonist at both the NMDA receptor and the L-type calcium channel. Our previous studies support NGP1-01. as a promising neuroprotective agent in diseases involving calcium-related excitotoxicity. We demonstrate here that NGP1-01 (1 and 10 mu M) pretreatment abrogates the effects of iron overload in brain endothelial cells protecting cellular viability. Both concentrations of NGP1-01 were found to attenuate iron-induced reduction in cellular viability to a similar extent, and were statistically significant. To further verify the mechanism, the L-type calcium channel agonist FPL 64176 was administered to promote iron uptake. Addition of NGP1-01 dose-dependently reduced FPL 64176 stimulated uptake of iron. These data support further evaluation of NGP1-01 as a neuroprotective agent, not only in diseases associated with excitotoxicity, but also in those of iron overload. (C) 2012 Elsevier B.V. All rights reserved.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1016/j.brainres.2012.10.029" target="_blank" rel="noreferrer noopener">10.1016/j.brainres.2012.10.029</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2012
activator
Allen D D
barrier
Brain research
Calcium Channels
channels provide
Geldenhuys W J
intracerebral hemorrhage
Iron-overload
Jones-Higgins M R
Journal Article or Conference Abstract Publication
Lockman J A
Neurodegeneration
neurodegenerative disorders
Neuroprotection
Neurosciences & Neurology
nimodipine
parkinsons-disease
Patrick J D
Permeability
rat-brain
toxicity
transport
Van der Schyf C J
Vascular endothelial cells
Voltage-gated calcium channel
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1038/sj.bmt.1700797" target="_blank" rel="noreferrer noopener">http://doi.org/10.1038/sj.bmt.1700797</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
1061-1064
Issue
11
Volume
19
Search for Full-text
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Dublin Core
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Title
A name given to the resource
Toxicity Associated With High-dose Cytosine Arabinoside And Total Body Irradiation As Conditioning For Allogeneic Bone Marrow Transplantation
Publisher
An entity responsible for making the resource available
Bone Marrow Transplantation
Date
A point or period of time associated with an event in the lifecycle of the resource
1997
1997-06
Subject
The topic of the resource
acute lymphoblastic-leukemia; adults; allogeneic; Biophysics; children; cyclophosphamide; cytosine; hematologic; Hematology; Immunology; lymphoma; malignancies; Oncology; preparative regimen; remission; survival; therapy; total body irradiation; toxicity; transplantation; transplantation
Creator
An entity primarily responsible for making the resource
Kumar M; Saleh A; Rao P V; Ochoa S; Meyers L; Miller A; GrahamPole J
Description
An account of the resource
Seventy-three patients with hematological cancers undergoing allogeneic bone marrow transplantation (BMT) were evaluated for event-free survival (EFS) and toxicity, All received 36 g/m(2) cytosine arabinoside (HDA) and 1200 cGy fractionated total body irradiation (TBI). We assessed the association of EFS and toxicities with the following risk factors: age, gender, diagnosis, initial relapse risk and patient-donor histocompatibility. The EFS probability is 33% at 800 days post-BMT, Twenty-six patients (36%) died of toxicity within 100 days and 14 (19%) have relapsed, EFS was inversely associated with age (P < 0.0001) and initial relapse risk (P = 0.007), The risk of pulmonary (P = 0.023) and hepatic toxicity (P = 0.011) increased with age, Diagnosis other than acute lymphoblastic leukemia (ALL) was a risk factor (P = 0.015) for graft-versushost disease (GVHD); and fewer ALL patients died from toxicity (P = 0.014), The probability of sepsis within 100 days post-BMT correlated (P = 0.007) with initial relapse risk. We conclude: (1) the lower EFS and greater pulmonary and hepatic toxicity associated with increasing age indicate a need for less toxic regimens that maintain high antileukemic efficacy for older patients; (2) the high GVHD and sepsis rates seen in certain categories of patients indicate a need for careful definition of eligibility criteria for this still highly toxic treatment.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1038/sj.bmt.1700797" target="_blank" rel="noreferrer noopener">10.1038/sj.bmt.1700797</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
1997
acute lymphoblastic-leukemia
adults
allogeneic
Biophysics
Bone Marrow Transplantation
Children
cyclophosphamide
cytosine
GrahamPole J
hematologic
Hematology
Immunology
Journal Article or Conference Abstract Publication
Kumar M
Lymphoma
malignancies
Meyers L
Miller A
Ochoa S
oncology
preparative regimen
Rao P V
Remission
Saleh A
Survival
therapy
total body irradiation
toxicity
Transplantation
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1177/106002809402800207" target="_blank" rel="noreferrer noopener">http://doi.org/10.1177/106002809402800207</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
195-196
Issue
2
Volume
28
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Title
A name given to the resource
Pronounced Metabolic Response To Modest Theophylline Overdose
Publisher
An entity responsible for making the resource available
Annals of Pharmacotherapy
Date
A point or period of time associated with an event in the lifecycle of the resource
1994
1994-02
Subject
The topic of the resource
intoxication; Pharmacology & Pharmacy; toxicity
Creator
An entity primarily responsible for making the resource
Hagley M T; Traeger S M; Schuckman H
Description
An account of the resource
OBJECTIVE: To describe a patient who developed significant metabolic abnormalities in response to a low-level theophylline ingestion. CASE SUMMARY: An 18-year-old man was examined after ingesting theophylline 3 g in a suicide attempt. Although his peak theophylline concentration was 157 mumol/L (28.2 mug/mL), it was associated with significant leukocytosis, hypokalemia, hypomagnesemia, hypophosphatemia, hyperglycemia, and lactic acidosis. These abnormalities have been previously associated with theophylline intoxication, but only in conjunction with much higher peak concentrations of theophylline. CONCLUSIONS: Significant metabolic abnormalities can occur with suicidal ingestion of relatively small amounts of theophylline. The presence of these abnormalities should be sought in theophylline overdoses. In the proper clinical circumstances, such abnormalities should raise suspicion of covert theophylline ingestion.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1177/106002809402800207" target="_blank" rel="noreferrer noopener">10.1177/106002809402800207</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
1994
Annals of Pharmacotherapy
Hagley M T
intoxication
Journal Article or Conference Abstract Publication
Pharmacology & Pharmacy
Schuckman H
toxicity
Traeger S M
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.2217/bmm.13.150" target="_blank" rel="noreferrer noopener">http://doi.org/10.2217/bmm.13.150</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
147-159
Issue
2
Volume
8
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Title
A name given to the resource
Targeted Liquid Chromatography-mass Spectrometry Analysis Of Serum Acylcarnitines In Acetaminophen Toxicity In Children
Publisher
An entity responsible for making the resource available
Biomarkers in Medicine
Date
A point or period of time associated with an event in the lifecycle of the resource
2014
2014-02
Subject
The topic of the resource
-oxidation; acetaminophen; acute liver-failure; acylcarnitine; biomarker; clinical; clofibrate; hepatic; hepatotoxicity; induced; induced hepatic-necrosis; metabolism; metabolomics; mice; multicenter; overdose; protein adducts; Research & Experimental Medicine; toxicity
Creator
An entity primarily responsible for making the resource
Bhattacharyya S; Yan K; Pence L; Simpson P M; Gill P; Letzig L G; Beger R D; Sullivan J E; Kearns G L; Reed M D; Marshall J D; Van Den Anker J N; James L P
Description
An account of the resource
Aim: Long-chain acylcarnitines have been postulated to be sensitive biomarkers of acetaminophen (APAP)-induced hepatotoxicity in mouse models. In the following study, the relationship of acylcarnitines with other known indicators of APAP toxicity was examined in children receiving low-dose (therapeutic) and high-dose (overdose' or toxic ingestion) exposure to APAP. Materials & methods: The study included three subject groups: group A (therapeutic dose, n = 187); group B (healthy controls, n = 23); and group C (overdose, n = 62). Demographic, clinical and laboratory data were collected for each subject. Serum samples were used for measurement of APAP protein adducts, a biomarker of the oxidative metabolism of APAP and for targeted metabolomics analysis of serum acylcarnitines using ultra performance liquid chromatography-triple-quadrupole mass spectrometry. Results: Significant increases in oleoyl- and palmitoyl-carnitines were observed with APAP exposure (low dose and overdose) compared with controls. Significant increases in serum ALT, APAP protein adducts and acylcarnitines were observed in overdose children that received delayed treatment (time to treatment from overdose >24 h) with the antidote N-acetylcysteine. Time to peak APAP protein adducts in serum was shorter than that of the acylcarnitines and serum ALT. Conclusion: Perturbations in long-chain acylcarnitines in children with APAP toxicity suggest that mitochrondrial injury and associated impairment in the -oxidation of fatty acids are clinically relevant as biomarkers of APAP toxicity.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.2217/bmm.13.150" target="_blank" rel="noreferrer noopener">10.2217/bmm.13.150</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
-oxidation
2014
ACETAMINOPHEN
acute liver-failure
acylcarnitine
Beger R D
Bhattacharyya S
Biomarker
Biomarkers in Medicine
Clinical
Clofibrate
Gill P
hepatic
hepatotoxicity
Induced
induced hepatic-necrosis
James L P
Journal Article or Conference Abstract Publication
Kearns G L
Letzig L G
Marshall J D
Metabolism
Metabolomics
Mice
multicenter
Overdose
Pence L
protein adducts
Reed M D
Research & Experimental Medicine
Simpson P M
Sullivan J E
toxicity
Van Den Anker J N
Yan K
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1002/1099-0496(200009)30:3%3C203::aid-ppul4%3E3.3.co;2-x" target="_blank" rel="noreferrer noopener">http://doi.org/10.1002/1099-0496(200009)30:3%3C203::aid-ppul4%3E3.3.co;2-x</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
203-206
Issue
3
Volume
30
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Title
A name given to the resource
Cystic Fibrosis Patients With And Without Central Nervous System Complications Following Lung Transplantation
Publisher
An entity responsible for making the resource available
Pediatric Pulmonology
Date
A point or period of time associated with an event in the lifecycle of the resource
2000
2000-09
Subject
The topic of the resource
CNS complications; convulsions; cyclosporin neurotoxicity; cyclosporine; cystic fibrosis; heart-transplantation; hyponatremia; liver; lung transplantation; methylprednisolone; neurologic complications; Pediatrics; receiving; recipients; Respiratory System; seizures; stroke; toxicity
Creator
An entity primarily responsible for making the resource
Goldstein A B; Goldstein L S; Perl M K; Haug M T; Arroliga A C; Stillwell P C
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1002/1099-0496(200009)30:3%3C203::aid-ppul4%3E3.3.co;2-x" target="_blank" rel="noreferrer noopener">10.1002/1099-0496(200009)30:3%3C203::aid-ppul4%3E3.3.co;2-x</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2000
Arroliga A C
CNS complications
Convulsions
cyclosporin neurotoxicity
cyclosporine
CYSTIC fibrosis
Goldstein A B
Goldstein L S
Haug M T
heart-transplantation
hyponatremia
Liver
lung transplantation
methylprednisolone
neurologic complications
Pediatric Pulmonology
Pediatrics
Perl M K
receiving
recipients
Respiratory System
Seizures
Stillwell P C
stroke
toxicity
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
n/a
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
159-173
Issue
1
Volume
9
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Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Scanning Electron-microscopy And Transmission Electron-microscopy Aspects Of Synergistic Antitumor-activity Of Vitamin-c Vitamin K-3 Combinations Against Human Prostatic-carcinoma Cells
Publisher
An entity responsible for making the resource available
Scanning Microscopy
Date
A point or period of time associated with an event in the lifecycle of the resource
1995
1995-03
Subject
The topic of the resource
cancer-chemotherapy; cultured-mammalian-cells; cytoskeleton; cytotoxicity; growth-invitro; induced oxidative stress; isolated hepatocytes; l-ascorbic-acid; lines; menadione; Microscopy; prostate carcinoma; ruthenium tetroxide; toxicity; ultrastructure; vitamin-c; vitamin-k-3
Creator
An entity primarily responsible for making the resource
Gilloteaux J; Jamison J M; Venugopal M; Giammar D; Summers J L
Identifier
An unambiguous reference to the resource within a given context
n/a
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
1995
cancer-chemotherapy
cultured-mammalian-cells
cytoskeleton
cytotoxicity
Giammar D
Gilloteaux J
growth-invitro
induced oxidative stress
isolated hepatocytes
Jamison J M
l-ascorbic-acid
lines
menadione
Microscopy
prostate carcinoma
ruthenium tetroxide
Scanning microscopy
Summers J L
toxicity
ultrastructure
Venugopal M
vitamin-c
vitamin-k-3
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1016/j.brachy.2007.01.006" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/j.brachy.2007.01.006</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
149-156
Issue
2
Volume
6
Search for Full-text
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Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Rectal Morbidity After Permanent Prostate Brachytherapy With Dose Escalation To Biologic Target Volumes Identified By Spect/ct Fusion
Publisher
An entity responsible for making the resource available
Brachytherapy
Date
A point or period of time associated with an event in the lifecycle of the resource
2007
2007-04
Subject
The topic of the resource
american brachytherapy; cancer; implantation; interstitial brachytherapy; Nuclear Medicine & Medical Imaging; Oncology; ProstaScint; prostate brachytherapy; quality-of-life; quality-of-life; radiation-therapy; radical prostatectomy; Radiology; radiotherapy; rectal morbidity; society recommendations; SPECT/CT; toxicity
Creator
An entity primarily responsible for making the resource
Ellis R J; Zhou H; Kaminsky D A; Fu P; Kim E Y; Sodee D B; Colussi V; Spirnak J P; Whalen C C; Resnick M I
Identifier
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<a href="http://doi.org/10.1016/j.brachy.2007.01.006" target="_blank" rel="noreferrer noopener">10.1016/j.brachy.2007.01.006</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
2007
american brachytherapy
brachytherapy
Cancer
Colussi V
Ellis R J
Fu P
implantation
interstitial brachytherapy
Kaminsky D A
Kim E Y
Nuclear Medicine & Medical Imaging
oncology
ProstaScint
prostate brachytherapy
quality-of-life
radiation-therapy
radical prostatectomy
Radiology
Radiotherapy
rectal morbidity
Resnick M I
society recommendations
Sodee D B
SPECT/CT
Spirnak J P
toxicity
Whalen C C
Zhou H
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
n/a
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
140-150
Volume
1139
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Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Sex Differences In Dopamine- And Vesicular Monoamine-transporter Functions Implications For Methamphetamine Use And Neurotoxicity
Publisher
An entity responsible for making the resource available
Drug Addiction: Research Frontiers and Treatment Advances
Date
A point or period of time associated with an event in the lifecycle of the resource
2008
2008
Subject
The topic of the resource
abuse; addiction; cd-1 mice; dopaminergic; drug abuse; estrogen; estrous-cycle; evoked striatal dopamine; female; gender-differences; gender-differences; inhibition; nigrostriatal; output; rats; sites; toxicity; uptake
Creator
An entity primarily responsible for making the resource
Dluzen D E; McDermott J L
Identifier
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n/a
Format
The file format, physical medium, or dimensions of the resource
Book Chapter
2008
abuse
Addiction
cd-1 mice
Dluzen D E
dopaminergic
Drug abuse
Drug Addiction: Research Frontiers and Treatment Advances
estrogen
estrous-cycle
evoked striatal dopamine
Female
gender-differences
inhibition
McDermott J L
nigrostriatal
output
Rats
sites
toxicity
uptake
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.1016/s0006-8993(96)00934-1" target="_blank" rel="noreferrer noopener">http://doi.org/10.1016/s0006-8993(96)00934-1</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
215-219
Issue
1
Volume
741
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Dublin Core
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Title
A name given to the resource
The Effects Of Intranasal Infusion Of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (mptp) Upon Catecholamine Concentrations Within Olfactory Bulbs And Corpus Striatum Of Male Mice
Publisher
An entity responsible for making the resource available
Brain Research
Date
A point or period of time associated with an event in the lifecycle of the resource
1996
1996-11
Subject
The topic of the resource
adult-rat; alzheimers-disease; brain; dopamine; epithelium; locus coeruleus; locus coeruleus; mouse; Neurosciences & Neurology; neurotoxicity; nigrostriatal; norepinephrine; parkinsons-disease; toxicity; transport; transporter; uptake
Creator
An entity primarily responsible for making the resource
Dluzen D E; Kefalas G
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1016/s0006-8993(96)00934-1" target="_blank" rel="noreferrer noopener">10.1016/s0006-8993(96)00934-1</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
1996
adult-rat
alzheimers-disease
Brain
Brain research
Department of Internal Medicine
Dluzen D E
Dopamine
Epithelium
Kefalas G
locus coeruleus
mouse
NEOMED College of Medicine
Neurosciences & Neurology
Neurotoxicity
nigrostriatal
Norepinephrine
parkinsons-disease
toxicity
transport
transporter
uptake
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
URL Address
<a href="http://doi.org/10.2217/bmm.13.150" target="_blank" rel="noreferrer noopener">http://doi.org/10.2217/bmm.13.150</a>
Rights
Article information provided for research and reference use only. All rights are retained by the journal listed under publisher and/or the creator(s).
Pages
147-159
Issue
2
Volume
8
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Dublin Core
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Title
A name given to the resource
Targeted Liquid Chromatography-mass Spectrometry Analysis Of Serum Acylcarnitines In Acetaminophen Toxicity In Children
Publisher
An entity responsible for making the resource available
Biomarkers in Medicine
Date
A point or period of time associated with an event in the lifecycle of the resource
2014
2014-02
Subject
The topic of the resource
-oxidation; acetaminophen; acute liver-failure; acylcarnitine; biomarker; clinical; clofibrate; hepatic; hepatotoxicity; induced; induced hepatic-necrosis; metabolism; metabolomics; mice; multicenter; overdose; protein adducts; Research & Experimental Medicine; toxicity
Creator
An entity primarily responsible for making the resource
Bhattacharyya S; Yan K; Pence L; Simpson P M; Gill P; Letzig L G; Beger R D; Sullivan J E; Kearns G L; Reed M D; Marshall J D; Van Den Anker J N; James L P
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.2217/bmm.13.150" target="_blank" rel="noreferrer noopener">10.2217/bmm.13.150</a>
Format
The file format, physical medium, or dimensions of the resource
Journal Article or Conference Abstract Publication
-oxidation
2014
ACETAMINOPHEN
acute liver-failure
acylcarnitine
Beger R D
Bhattacharyya S
Biomarker
Biomarkers in Medicine
Clinical
Clofibrate
Gill P
hepatic
hepatotoxicity
Induced
induced hepatic-necrosis
James L P
Kearns G L
Letzig L G
Marshall J D
Metabolism
Metabolomics
Mice
multicenter
Overdose
Pence L
protein adducts
Reed M D
Research & Experimental Medicine
Simpson P M
Sullivan J E
toxicity
Van Den Anker J N
Yan K