Browse Items · NEOMED Bibliography Database

Tamoxifen (tmx) Mimics The Vascular Effects Of Estrogen (e-2) By Upregulating Constrictor Prostanoid Function By An E-2 Receptor-dependent Mechanism

Ovariectomy Abolishes Constrictor Prostanoid-potentiated Contractions To Vasopressin (vp) In Female Rat Aorta

Constrictor Prostanoids Contribute To Sexual Dimorphism In Vasopressin (vp)-induced Contraction Of Rat Aorta

Long-term Tamoxifen (tmx)-treatment Enhances Endothelium-dependent Nitric Oxide (no) And Constrictor Prostanoid (pg) Function In Female Rat Aorta

Estrogen or selective estrogen receptor modulators (SERMs) inhibit Ang II-induced arachidonic acid (AA) release and growth in vascular smooth muscle cells (VSMC)

Androgen-receptor defect abolishes sex differences in nitric oxide and reactivity to vasopressin in rat aorta

Contractions of rat thoracic aorta to vasopressin WP) are threefold higher in females (F) than in males (M), primarily because nitric oxide (NO) attenuation of contraction is greater in M. To determine the role of the androgen receptor (AR) in this…

Browse Items (6 total)

Tamoxifen (tmx) Mimics The Vascular Effects Of Estrogen (e-2) By Upregulating Constrictor Prostanoid Function By An E-2 Receptor-dependent Mechanism

Ovariectomy Abolishes Constrictor Prostanoid-potentiated Contractions To Vasopressin (vp) In Female Rat Aorta

Constrictor Prostanoids Contribute To Sexual Dimorphism In Vasopressin (vp)-induced Contraction Of Rat Aorta

Long-term Tamoxifen (tmx)-treatment Enhances Endothelium-dependent Nitric Oxide (no) And Constrictor Prostanoid (pg) Function In Female Rat Aorta

Estrogen or selective estrogen receptor modulators (SERMs) inhibit Ang II-induced arachidonic acid (AA) release and growth in vascular smooth muscle cells (VSMC)

Androgen-receptor defect abolishes sex differences in nitric oxide and reactivity to vasopressin in rat aorta