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Mitochondrial dysfunction triggers catabolic response in chondrocytes via ROS mediated activation of JNK/AP1 pathway.

Mitochondrial function is impaired in osteoarthritis (OA) but its impact on cartilage catabolism is not fully understood. Here, we investigated the molecular mechanism of mitochondrial dysfunction-induced activation of catabolic response in …

Tags: 2020, Ahmad N, Ansari M Y, cFos-AP1, Chondrocytes, Department of Anatomy & Neurobiology, Haqqi TM, Inflammation, JNK, Journal of Cell Science, journalArticle, NEOMED College of Medicine, NEOMED Student Publications, Novak K, October 2020 List, Osteoarthritis, Redox, Voleti S, Wase SJ

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Browse Items (2 total)

Mitochondrial dysfunction triggers catabolic response in chondrocytes via ROS mediated activation of JNK/AP1 pathway.

MITOCHONDRIAL DYSFUNCTION IN OSTEOARTHRITIS AND AGED CARTILAGE TRIGGERS INFLAMMATORY RESPONSE AND MATRIX DEGRADATION VIA ROS MEDIATED ACTIVATION OF JNK-MAPK/CFOS-AP1 AXIS IN CHONDROCYTES